Journal
JOURNAL OF TRAUMATIC STRESS
Volume 22, Issue 5, Pages 416-426Publisher
WILEY-BLACKWELL
DOI: 10.1002/jts.20435
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Funding
- NIMH NIH HHS [K08 MH070627-06, T32 MH078788, R01 MH078928-02, K08 MH070627, F32 MH083469, F32 MH083469-01, R01 MH078928, MH078928, L40 MH084279-01, K01 MH087240, L40 MH084279] Funding Source: Medline
- Wellcome Trust [083469] Funding Source: Medline
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The authors provide a detailed review of the extant gene-environment interaction (GxE) research in the etiology of posttraumatic stress disorder (PTSD). They begin with a discussion of why PTSD is uniquely fitting for the innovative framework of GxE methodology, followed by a review of the heritability and main effect molecular genetics studies of PTSD. Next, they discuss the six GxE investigations to date on PTSD. They end with a discussion of future directions and significance of this research, with an emphasis on the expansion of psychosocial factors that may befitting environmental variables for inclusion in this new research area. The authors posit that GxE research is vital to elucidating risk and resilience following exposure to a potentially traumatic event.
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