4.5 Article

NaV1.9 Potentiates Oxidized Phospholipid-Induced TRP Responses Only under Inflammatory Conditions

Related references

Note: Only part of the references are listed.
Article Medicine, Research & Experimental

Sodium channel NaV1.9 mutations associated with insensitivity to pain dampen neuronal excitability

Jianying Huang et al.

JOURNAL OF CLINICAL INVESTIGATION (2017)

Review Physiology

Neurobiology of local and intercellular BDNF signaling

Manju Sasi et al.

PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY (2017)

Article Multidisciplinary Sciences

Inflammatory pain control by blocking oxidized phospholipid-mediated TRP channel activation

Beatrice Oehler et al.

SCIENTIFIC REPORTS (2017)

Review Immunology

The regulation of inflammation by oxidized phospholipids

Stefan Freigang

EUROPEAN JOURNAL OF IMMUNOLOGY (2016)

Review Multidisciplinary Sciences

Pain regulation by non-neuronal cells and inflammation

Ru-Rong Ji et al.

SCIENCE (2016)

Article Neurosciences

NaV 1.9: a sodium channel linked to human pain

Sulayman D. Dib-Hajj et al.

NATURE REVIEWS NEUROSCIENCE (2015)

Letter Biochemistry & Molecular Biology

The phenotype of congenital insensitivity to pain due to the NaV1.9 variant p.L811P

Christopher Geoffrey Woods et al.

EUROPEAN JOURNAL OF HUMAN GENETICS (2015)

Article Clinical Neurology

Gain-of-function mutations in sodium channel NaV1.9 in painful neuropathy

Jianying Huang et al.

BRAIN (2014)

Review Clinical Neurology

Painful and painless channelopathies

David L. H. Bennett et al.

LANCET NEUROLOGY (2014)

Review Neurosciences

Regulating excitability of peripheral afferents: emerging ion channel targets

Stephen G. Waxman et al.

NATURE NEUROSCIENCE (2014)

Review Biotechnology & Applied Microbiology

Emerging targets in neuroinflammation-driven chronic pain

Ru-Rong Ji et al.

NATURE REVIEWS DRUG DISCOVERY (2014)

Article Multidisciplinary Sciences

TRPA1 channels mediate acute neurogenic inflammation and pain produced by bacterial endotoxins

Victor Meseguer et al.

NATURE COMMUNICATIONS (2014)

Article Genetics & Heredity

Gain-of-Function Mutations in SCN11A Cause Familial Episodic Pain

Xiang Yang Zhang et al.

AMERICAN JOURNAL OF HUMAN GENETICS (2013)

Review Cell Biology

TRP Channels and Pain

David Julius

ANNUAL REVIEW OF CELL AND DEVELOPMENTAL BIOLOGY, VOL 29 (2013)

Article Biochemistry & Molecular Biology

Cell-autonomous axon growth of young motoneurons is triggered by a voltage-gated sodium channel

Andrea Wetzel et al.

CHANNELS (2013)

Article Physiology

Mechanism of sodium channel NaV1.9 potentiation by G-protein signaling

Carlos G. Vanoye et al.

JOURNAL OF GENERAL PHYSIOLOGY (2013)

Article Neurosciences

Effect of cholesterol depletion on the pore dilation of TRPV1

Erik T. Jansson et al.

MOLECULAR PAIN (2013)

Article Genetics & Heredity

A de novo gain-of-function mutation in SCN11A causes loss of pain perception

Enrico Leipold et al.

NATURE GENETICS (2013)

Review Biochemistry & Molecular Biology

Protein modification by aldehydophospholipids and its functional consequences

Ute Stemmer et al.

BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES (2012)

Article Biochemistry & Molecular Biology

Role of Nav1.9 in activity-dependent axon growth in motoneurons

Narayan Subramanian et al.

HUMAN MOLECULAR GENETICS (2012)

Article Biochemical Research Methods

NIH Image to ImageJ: 25 years of image analysis

Caroline A. Schneider et al.

NATURE METHODS (2012)

Article Neurosciences

Imaging Calcium in Neurons

Christine Grienberger et al.

NEURON (2012)

Review Neurosciences

Sodium Channels in Normal and Pathological Pain

Sulayman D. Dib-Hajj et al.

ANNUAL REVIEW OF NEUROSCIENCE, VOL 33 (2010)

Review Biochemistry & Molecular Biology

Generation and Biological Activities of Oxidized Phospholipids

Valery N. Bochkov et al.

ANTIOXIDANTS & REDOX SIGNALING (2010)

Review Medicine, Research & Experimental

Nociceptors: the sensors of the pain pathway

Adrienne E. Dubin et al.

JOURNAL OF CLINICAL INVESTIGATION (2010)

Review Biochemistry & Molecular Biology

Cellular and Molecular Mechanisms of Pain

Allan I. Basbaum et al.

Article Biochemical Research Methods

Transfection of rat or mouse neurons by biolistics or electroporation

Sulayman D. Dib-Hajj et al.

NATURE PROTOCOLS (2009)

Review Neurosciences

Regulation of firing frequency in nociceptive neurons by pro-inflammatory mediators

Aliakmal Momin et al.

EXPERIMENTAL BRAIN RESEARCH (2009)

Article Biochemistry & Molecular Biology

Anti-inflammatory apoA-I-mimetic peptides bind oxidized lipids with much higher affinity than human apoA-I

Brian J. Van Lenten et al.

JOURNAL OF LIPID RESEARCH (2008)

Article Neurosciences

GTP up-regulated persistent Na+ current and enhanced nociceptor excitability require NaV1.9

Johan A. R. Ostman et al.

JOURNAL OF PHYSIOLOGY-LONDON (2008)

Review Biochemistry & Molecular Biology

Oxidized phospholipids: From molecular properties to disease

Gilbert O. Fruhwirth et al.

BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2007)

Article Multidisciplinary Sciences

Noxious compounds activate TRPA1 ion channels through covalent modification of cysteines

Lindsey J. Macpherson et al.

NATURE (2007)

Article Multidisciplinary Sciences

TRP channel activation by reversible covalent modification

Andrew Hinman et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)

Article Biochemistry & Molecular Biology

TRPA1 mediates the inflammatory actions of environmental irritants and proalgesic agents

DM Bautista et al.

Article Multidisciplinary Sciences

Contribution of the tetrodotoxin-resistant voltage-gated sodium channel Nav1.9 to sensory transmission and nociceptive behavior

BT Priest et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2005)

Article Hematology

Oxidized phospholipids trigger atherogenic inflammation in murine arteries

A Furnkranz et al.

ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY (2005)

Article Multidisciplinary Sciences

Neurotrophin-evoked depolarization requires the sodium channel NaV1.9

R Blum et al.

NATURE (2002)

Article Multidisciplinary Sciences

Impaired nociception and pain sensation in mice lacking the capsaicin receptor

MJ Caterina et al.

SCIENCE (2000)