Related references
Note: Only part of the references are listed.Long-term depression in Purkinje neurons is persistently impaired following cardiac arrest and cardiopulmonary resuscitation in mice
Nidia Quillinan et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2017)
Autonomous CaMKII Activity as a Drug Target for Histological and Functional Neuroprotection after Resuscitation from Cardiac Arrest
Guiying Deng et al.
CELL REPORTS (2017)
THERAPEUTIC HYPOTHERMIA PROTECTS AGAINST ISCHEMIA-INDUCED IMPAIRMENT OF SYNAPTIC PLASTICITY FOLLOWING JUVENILE CARDIAC ARREST IN SEX-DEPENDENT MANNER
R. M. Dietz et al.
NEUROSCIENCE (2016)
Status Epilepticus Enhances Depotentiation after Fully Established LTP in an NMDAR-Dependent but GluN2B-Independent Manner
Xiati Guli et al.
NEURAL PLASTICITY (2016)
Cognitive Function in Survivors of Out-of-Hospital Cardiac Arrest After Target Temperature Management at 33°C Versus 36°C
Gisela Lilja et al.
CIRCULATION (2015)
Ischemic LTP: NMDA-dependency and dorso/ventral distribution within the hippocampus
Nicola Maggio et al.
HIPPOCAMPUS (2015)
Increasing small conductance Ca2+-activated potassium channel activity reverses ischemia-induced impairment of long-term potentiation
J. E. Orfila et al.
EUROPEAN JOURNAL OF NEUROSCIENCE (2014)
A novel mouse model of pediatric cardiac arrest and cardiopulmonary resuscitation reveals age-dependent neuronal sensitivities to ischemic injury
G. Deng et al.
JOURNAL OF NEUROSCIENCE METHODS (2014)
ACETYL-L-CARNITINE NORMALIZES THE IMPAIRED LONG-TERM POTENTIATION AND SPINE DENSITY IN A RAT MODEL OF GLOBAL ISCHEMIA
K. Kocsis et al.
NEUROSCIENCE (2014)
Active Calcium/Calmodulin-Dependent Protein Kinase II (CaMKII) Regulates NMDA Receptor Mediated Postischemic Long-Term Potentiation (i-LTP) by Promoting the Interaction between CaMKII and NMDA Receptors in Ischemia
Ning Wang et al.
NEURAL PLASTICITY (2014)
Switching off LTP: mGlu and NMDA Receptor-Dependent Novelty Exploration-Induced Depotentiation in the Rat Hippocampus
Yingjie Qi et al.
CEREBRAL CORTEX (2013)
Kainate postconditioning restores LTP in ischemic hippocampal CA1: Onset-dependent second pathophysiological stress
David Nagy et al.
NEUROPHARMACOLOGY (2011)
Normothermic Cardiac Arrest and Cardiopulmonary Resuscitation: A Mouse Model of Ischemia-Reperfusion Injury
Michael P. Hutchens et al.
JOVE-JOURNAL OF VISUALIZED EXPERIMENTS (2011)
CaMKII Autonomy Is Required for Initiating But Not for Maintaining Neuronal Long-Term Information Storage
Isabelle Buard et al.
JOURNAL OF NEUROSCIENCE (2010)
Plasticity and repair in the post-ischemic brain
Massimiliano Di Filippo et al.
NEUROPHARMACOLOGY (2008)
Cognitive and neurophysiological outcome of cardiac arrest survivors treated with therapeutic hypothermia
Marjaana Tiainen et al.
STROKE (2007)
Neurosteroid estradiol rescues ischemia-induced deficit in the long-term potentiation, of rat hippocampal CA1 neurons
Xiaoniu Dai et al.
NEUROPHARMACOLOGY (2007)
Glutamate receptor changes associated with transient anoxia/hypoglycaemia in hippocampal slice cultures
P Quintana et al.
EUROPEAN JOURNAL OF NEUROSCIENCE (2006)
Effects of hypothermia for a short period on histologic outcome and extracellular glutamate concentration during and after cardiac arrest in rats
K Takata et al.
CRITICAL CARE MEDICINE (2005)
Histopathological and behavioral characterization of a novel model of cardiac arrest and cardiopulmonary resuscitation in mice
J Kofler et al.
JOURNAL OF NEUROSCIENCE METHODS (2004)
Cardiac arrest with cardiopulmonary resuscitation reduces dendritic spine density in CA1 pyramidal cells and selectively alters acquisition of spatial memory
GN Neigh et al.
EUROPEAN JOURNAL OF NEUROSCIENCE (2004)
Synaptic plasticity in the ischaemic brain
P Calabresi et al.
LANCET NEUROLOGY (2003)
Characterization of the mechanism underlying the reversal of long term potentiation by low frequency stimulation at hippocampal CA1 synapses
CC Huang et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Acute decrease in net glutamate uptake during energy deprivation
D Jabaudon et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)
Glutamate release in severe brain ischaemia is mainly by reversed uptake
DJ Rossi et al.
NATURE (2000)