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15-Epi-lipoxin A4 Inhibits Myeloperoxidase Signaling and Enhances Resolution of Acute Lung Injury
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Serum amyloid A is a biomarker of acute exacerbations of chronic obstructive pulmonary disease
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Synthetic chemerin-derived peptides suppress inflammation through ChemR23
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Aspirin-triggered lipoxins override the apoptosis-delaying action of serum amyloid A in human neutrophils:: A novel mechanism for resolution of inflammation
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Resolvin E1 and protectin D1 activate inflammation-resolution programmes
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Resolvin E1 selectively interacts with leukotriene B4 receptor BLT1 and ChemR23 to regulate inflammation
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Posttreatment with aspirin-triggered lipoxin A4 analog attenuates lipopolysaccharide-induced acute lung injury in mice:: The role of heme oxygenase-1
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Protectin D1 is generated in asthma and dampens airway inflammation and hyperresponsiveness
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Lipoxin A4 regulates bronchial epithelial cell responses to acid injury
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Aspirin-triggered lipoxin A4 and B4 analogs block extracellular signal-regulated kinase-dependent TNF-α secretion from human T cells
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