Related references
Note: Only part of the references are listed.Prometastatic mechanisms of CAF-mediated EMT regulation in pancreatic cancer cells
Tao Shan et al.
INTERNATIONAL JOURNAL OF ONCOLOGY (2017)
Limited heterogeneity of known driver gene mutations among the metastases of individual patients with pancreatic cancer
Alvin P. Makohon-Moore et al.
NATURE GENETICS (2017)
Epigenomic reprogramming during pancreatic cancer progression links anabolic glucose metabolism to distant metastasis
Oliver G. McDonald et al.
NATURE GENETICS (2017)
EMT, CSCs, and drug resistance: the mechanistic link and clinical implications
Tsukasa Shibue et al.
NATURE REVIEWS CLINICAL ONCOLOGY (2017)
The influence of neural invasion on survival and tumor recurrence in pancreatic ductal adenocarcinoma - A systematic review and meta-analysis
Stephan Schorn et al.
SURGICAL ONCOLOGY-OXFORD (2017)
Pain in Patients with Pancreatic Cancer: Prevalence, Mechanisms, Management and Future Developments
Andreas I. Koulouris et al.
DIGESTIVE DISEASES AND SCIENCES (2017)
IL-6/STAT3 Plays a Regulatory Role in the Interaction Between Pancreatic Stellate Cells and Cancer Cells
Shin Hamada et al.
DIGESTIVE DISEASES AND SCIENCES (2016)
Prrx1 isoform switching regulates pancreatic cancer invasion and metastatic colonization
Shigetsugu Takano et al.
GENES & DEVELOPMENT (2016)
Morphological heterogeneity in ductal adenocarcinoma of the pancreas - Does it matter?
Caroline Verbeke
PANCREATOLOGY (2016)
Differential membranous E-cadherin expression, cell proliferation and O-GlcNAcylation between primary and metastatic nodal lesion in colorectal cancer
Tae Jung Jang
PATHOLOGY RESEARCH AND PRACTICE (2016)
Tumor Budding Is an Independent Adverse Prognostic Factor in Pancreatic Ductal Adenocarcinoma
Kate O'Connor et al.
AMERICAN JOURNAL OF SURGICAL PATHOLOGY (2015)
Expression of E-cadherin repressors SNAIL, ZEB1 and ZEB2 by tumour and stromal cells influences tumour-budding phenotype and suggests heterogeneity of stromal cells in pancreatic cancer
J. A. Galvan et al.
BRITISH JOURNAL OF CANCER (2015)
Upregulation of integrin β4 promotes epithelial-mesenchymal transition and is a novel prognostic marker in pancreatic ductal adenocarcinoma
Y. Masugi et al.
LABORATORY INVESTIGATION (2015)
Neural Regulation of Hematopoiesis, Inflammation, and Cancer
Maher Hanoun et al.
NEURON (2015)
Accumulation of FOXP3+T-cells in the tumor microenvironment is associated with an epithelial-mesenchymal-transition-type tumor budding phenotype and is an independent prognostic factor in surgically resected pancreatic ductal adenocarcinoma
Martin Wartenberg et al.
ONCOTARGET (2015)
Cancer cell invasion and EMT marker expression: a three-dimensional study of the human cancer-host interface
P. Bronsert et al.
JOURNAL OF PATHOLOGY (2014)
Upregulated SMAD3 promotes epithelial-mesenchymal transition and predicts poor prognosis in pancreatic ductal adenocarcinoma
Ken Yamazaki et al.
LABORATORY INVESTIGATION (2014)
Tumour budding is a strong and independent prognostic factor in pancreatic cancer
E. Karamitopoulou et al.
EUROPEAN JOURNAL OF CANCER (2013)
Loss of Raf-1 kinase inhibitor protein (RKIP) is strongly associated with high-grade tumor budding and correlates with an aggressive phenotype in pancreatic ductal adenocarcinoma (PDAC)
Eva Karamitopoulou et al.
JOURNAL OF TRANSLATIONAL MEDICINE (2013)
EMT and MET in Metastasis: Where Are the Cancer Stem Cells?
Thomas Brabletz
CANCER CELL (2012)
EMT and Dissemination Precede Pancreatic Tumor Formation
Andrew D. Rhim et al.
CELL (2012)
Smad phosphoisoform signaling specificity: the right place at the right time
Koichi Matsuzaki
CARCINOGENESIS (2011)
Intrapancreatic Nerve Invasion as a Predictor for Recurrence After Pancreaticoduodenectomy in Patients With Invasive Ductal Carcinoma of the Pancreas
Kazuaki Shimada et al.
PANCREAS (2011)
Solitary cell infiltration is a novel indicator of poor prognosis and epithelial-mesenchymal transition in pancreatic cancer
Yohei Masugi et al.
HUMAN PATHOLOGY (2010)
Synuclein-γ Is Closely Involved in Perineural Invasion and Distant Metastasis in Mouse Models and Is a Novel Prognostic Factor in Pancreatic Cancer
Taizo Hibi et al.
CLINICAL CANCER RESEARCH (2009)
MUC1 is a counter-receptor for myelin-associated glycoprotein (Siglec-4a) and their interaction contributes to adhesion in pancreatic cancer perineural invasion
Benjamin J. Swanson et al.
CANCER RESEARCH (2007)
Co-culturing human prostate carcinoma cells with hepatocytes leads to increased expression of E-cadherin
C. C. Yates et al.
BRITISH JOURNAL OF CANCER (2007)
Establishment of perineural invasion models and analysis of gene expression revealed an invariant chain (CD74) as a possible molecule involved in perineural invasion in pancreatic cancer
N Koide et al.
CLINICAL CANCER RESEARCH (2006)
A proposal for a new and more practical grading scheme for pancreatic ductal adenocarcinoma
NV Adsay et al.
AMERICAN JOURNAL OF SURGICAL PATHOLOGY (2005)
Elevated expression of E-cadherin and α-, and γ-catenins in metastatic lesions compared with primary epithelial ovarian carcinomas
T Imai et al.
HUMAN PATHOLOGY (2004)
Dysadherin overexpression in pancreatic ductal adenocarcinoma reflects tumor aggressiveness: Relationship to E-cadherin expression
T Shimamura et al.
JOURNAL OF CLINICAL ONCOLOGY (2003)
E-cadherin expression in primary carcinomas of the breast and its distant metastases
PJ Kowalski et al.
BREAST CANCER RESEARCH (2003)
E-cadherin expression in prostate cancer: A broad survey using high-density tissue microarray technology
MA Rubin et al.
HUMAN PATHOLOGY (2001)