Related references
Note: Only part of the references are listed.VPS35, the Retromer Complex and Parkinson's Disease
Erin T. Williams et al.
JOURNAL OF PARKINSONS DISEASE (2017)
Parkinson's disease-associated mutant VPS35 causes mitochondrial dysfunction by recycling DLP1 complexes
Wenzhang Wang et al.
NATURE MEDICINE (2016)
Genetics in Parkinson disease: Mendelian versus non-Mendelian inheritance
Dena G. Hernandez et al.
JOURNAL OF NEUROCHEMISTRY (2016)
VPS35 Deficiency or Mutation Causes Dopaminergic Neuronal Loss by Impairing Mitochondrial Fusion and Function
Fu-Lei Tang et al.
CELL REPORTS (2015)
VPS35-deficiency results in an impaired AMPA receptor trafficking and decreased dendritic spine maturation
Yun Tian et al.
MOLECULAR BRAIN (2015)
Retromer-dependent neurotransmitter receptor trafficking to synapses is altered by the Parkinson's disease VPS35 mutation p.D620N
L. N. Munsie et al.
HUMAN MOLECULAR GENETICS (2015)
Retromer Binding to FAM21 and the WASH Complex Is Perturbed by the Parkinson Disease-Linked VPS35(D620N) Mutation
Ian J. McGough et al.
CURRENT BIOLOGY (2014)
Parkinson's disease-linked mutations in VPS35 induce dopaminergic neurodegeneration
Elpida Tsika et al.
HUMAN MOLECULAR GENETICS (2014)
Mutation in VPS35 associated with Parkinson's disease impairs WASH complex association and inhibits autophagy
Eszter Zavodszky et al.
NATURE COMMUNICATIONS (2014)
In vivo evidence of pathogenicity of VPS35 mutations in the Drosophila
Hua-shan Wang et al.
MOLECULAR BRAIN (2014)
A Mutation in VPS35, Encoding a Subunit of the Retromer Complex, Causes Late-Onset Parkinson Disease
Alexander Zimprich et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2011)
VPS35 Mutations in Parkinson Disease
Carles Vilarino-Gueell et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2011)
Share Paper
