Journal
CANCER CELL
Volume 33, Issue 6, Pages 949-951Publisher
CELL PRESS
DOI: 10.1016/j.ccell.2018.05.010
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Funding
- NIH [5R35CA197532-02]
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To avoid reactive oxygen species (ROS)-induced cell death, cancer cells increase their antioxidant defense system. In this issue of Cancer Cell, Takahashi et al. identify a novel, non-canonical oxidative stress defense mechanism involving TRPA1, a redox-sensitive Ca2+ channel, and the upregulation of anti-apoptotic pathways to promote cancer cell survival.
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