Journal
AUTOPHAGY
Volume 14, Issue 2, Pages 243-251Publisher
TAYLOR & FRANCIS INC
DOI: 10.1080/15548627.2017.1402992
Keywords
Autophagy; microbes mitochondria; inflamamsome; interferon; lysosome; phagosome
Categories
Funding
- HHS \ NIH \ National Institute of Allergy and Infectious Diseases (NIAID) [AI42999, AI109725, AI111935]
- HHS \ NIH \ National Institute of General Medical Sciences (NIGMS) [GM121176]
- NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES [U19AI109725, R37AI042999] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF GENERAL MEDICAL SCIENCES [P20GM121176] Funding Source: NIH RePORTER
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Macroautophagy/autophagy is a homeostatic process with multiple effects on immunity. One of the pivotal contributions of autophagy in immunity is the cell autonomous control of inflammation. This property leads to systemic consequences and thereby influences the development of innate and adaptive immunity, which promotes or suppresses pathology in various disease contexts. In this review we focus on the intersections between autophagy and inflammasome activation, autophagy and interferons, and autophagy and inflammation in association with infection.
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