4.8 Article

Complement C3 deficiency protects against neurodegeneration in aged plaque-rich APP/PS1 mice

Related references

Note: Only part of the references are listed.
Review Immunology

Inflammation in Alzheimer's disease: Lessons learned from microglia-depletion models

Elizabeth E. Spangenberg et al.

BRAIN BEHAVIOR AND IMMUNITY (2017)

Article Multidisciplinary Sciences

Novel allele-dependent role for APOE in controlling the rate of synapse pruning by astrocytes

Won-Suk Chung et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2016)

Article Multidisciplinary Sciences

Complement and microglia mediate early synapse loss in Alzheimer mouse models

Soyon Hong et al.

SCIENCE (2016)

Article Neurosciences

Complement C3-Deficient Mice Fail to Display Age-Related Hippocampal Decline

Qiaoqiao Shi et al.

JOURNAL OF NEUROSCIENCE (2015)

Article Neurosciences

Altered cognitive performance and synaptic function in the hippocampus of mice lacking C3

Marta Perez-Alcazar et al.

EXPERIMENTAL NEUROLOGY (2014)

Article Neurosciences

Neurodegeneration by Activation of the Microglial Complement-Phagosome Pathway

Liviu-Gabriel Bodea et al.

JOURNAL OF NEUROSCIENCE (2014)

Review Clinical Neurology

Microglial priming in neurodegenerative disease

V. Hugh Perry et al.

NATURE REVIEWS NEUROLOGY (2014)

Article Biochemistry & Molecular Biology

Microglia Promote Learning-Dependent Synapse Formation through Brain-Derived Neurotrophic Factor

Christopher N. Parkhurst et al.

Article Immunology

A quantitative spatiotemporal analysis of microglia morphology during ischemic stroke and reperfusion

Helena W. Morrison et al.

JOURNAL OF NEUROINFLAMMATION (2013)

Article Clinical Neurology

Synaptic Changes in the Dentate Gyrus of APP/PS1 Transgenic Mice Revealed by Electron Microscopy

Lidia Alonso-Nanclares et al.

JOURNAL OF NEUROPATHOLOGY AND EXPERIMENTAL NEUROLOGY (2013)

Article Neurosciences

A Dramatic Increase of C1q Protein in the CNS during Normal Aging

Alexander H. Stephan et al.

JOURNAL OF NEUROSCIENCE (2013)

Article Neurosciences

TGF-β signaling regulates neuronal Clq expression and developmental synaptic refinement

Allison R. Bialas et al.

NATURE NEUROSCIENCE (2013)

Article Multidisciplinary Sciences

C3-dependent mechanism of microglial priming relevant to multiple sclerosis

Valeria Ramaglia et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2012)

Article Behavioral Sciences

Hippocampus-dependent place learning enables spatial flexibility in C57BL6/N mice

Karl R. Kleinknecht et al.

FRONTIERS IN BEHAVIORAL NEUROSCIENCE (2012)

Article Clinical Neurology

Histological and Direct Evidence for the Role of Complement in the Neuroinflammation of AD

Robert Veerhuis

CURRENT ALZHEIMER RESEARCH (2011)

Article Pharmacology & Pharmacy

Complement receptor 3 (CD11b/CD18) is implicated in the elimination of β-amyloid peptides

Nada Choucair-Jaafar et al.

FUNDAMENTAL & CLINICAL PHARMACOLOGY (2011)

Review Biochemistry & Molecular Biology

Complement in the brain

Robert Veerhuis et al.

MOLECULAR IMMUNOLOGY (2011)

Article Multidisciplinary Sciences

Soluble amyloid β-protein dimers isolated from Alzheimer cortex directly induce Tau hyperphosphorylation and neuritic degeneration

Ming Jin et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2011)

Article Multidisciplinary Sciences

Quantifying Synapses: an Immunocytochemistry-based Assay to Quantify Synapse Number

Dominic M. Ippolito et al.

Jove-Journal of Visualized Experiments (2010)

Article Neurosciences

Widespread Changes in Dendritic Spines in a Model of Alzheimer's Disease

S. Knafo et al.

CEREBRAL CORTEX (2009)

Article Geriatrics & Gerontology

Amyloid precursor protein increases cortical neuron size in transgenic mice

Esther S. Oh et al.

NEUROBIOLOGY OF AGING (2009)

Review Geriatrics & Gerontology

Synaptic contact number and size in stratum radiatum CA1 of APP/PS1ΔE9 transgenic mice

Mark J. West et al.

NEUROBIOLOGY OF AGING (2009)

Article Multidisciplinary Sciences

Oligomeric amyloid β associates with postsynaptic densities and correlates with excitatory synapse loss near senile plaques

Robert M. Koffie et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)

Article Biochemistry & Molecular Biology

Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer's disease

Jun Zhou et al.

JOURNAL OF NEUROCHEMISTRY (2008)

Article Neurosciences

Molecular signatures of neurodegeneration in the cortex of PS1/PS2 double knockout mice

Karoly Mirnics et al.

MOLECULAR NEURODEGENERATION (2008)

Article Biochemistry & Molecular Biology

Age-related alterations of Nestin-immunoreactive neurons in rat basal forebrain with aged memory deficit

Dongpei Li et al.

NEUROCHEMISTRY INTERNATIONAL (2008)

Article Biochemistry & Molecular Biology

The classical complement cascade mediates CNS synapse elimination

Beth Stevens et al.

Article Biochemistry & Molecular Biology

Reduced β-amyloid production and increased inflammatory responses in presenilin conditional knock-out mice

V Beglopoulos et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2004)

Article Multidisciplinary Sciences

Alzheimer's disease is a synaptic failure

DJ Selkoe

SCIENCE (2002)

Article Multidisciplinary Sciences

Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice

T Wyss-Coray et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)

Article Pathology

Inflammatory responses to amyloidosis in a transgenic mouse model of Alzheimer's disease

Y Matsuoka et al.

AMERICAN JOURNAL OF PATHOLOGY (2001)

Review Geriatrics & Gerontology

Inflammation and Alzheimer's disease

H Akiyama et al.

NEUROBIOLOGY OF AGING (2000)