Related references
Note: Only part of the references are listed.RIPK3 Activates Parallel Pathways of MLKL-Driven Necroptosis and FADD-Mediated Apoptosis to Protect against Influenza A Virus
Shoko Nogusa et al.
CELL HOST & MICROBE (2016)
ZBP1/DAI is an innate sensor of influenza virus triggering the NLRP3 inflammasome and programmed cell death pathways
Teneema Kuriakose et al.
SCIENCE IMMUNOLOGY (2016)
Herpes Simplex Virus Suppresses Necroptosis in Human Cells
Hongyan Guo et al.
CELL HOST & MICROBE (2015)
RIP1/RIP3 Binding to HSV-1 ICP6 Initiates Necroptosis to Restrict Virus Propagation in Mice
Zhe Huang et al.
CELL HOST & MICROBE (2015)
Suppression of RIP3-dependent Necroptosis by Human Cytomegalovirus
Shinya Omoto et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2015)
Manipulation of apoptosis and necroptosis signaling by herpesviruses
Hongyan Guo et al.
MEDICAL MICROBIOLOGY AND IMMUNOLOGY (2015)
Necroptosis: The Trojan horse in cell autonomous antiviral host defense
Edward S. Mocarski et al.
VIROLOGY (2015)
Distinct roles of RIP1-RIP3 hetero- and RIP3-RIP3 homo-interaction in mediating necroptosis
X-N Wu et al.
CELL DEATH AND DIFFERENTIATION (2014)
Mixed Lineage Kinase Domain-like Protein MLKL Causes Necrotic Membrane Disruption upon Phosphorylation by RIP3
Huayi Wang et al.
MOLECULAR CELL (2014)
Staying Alive: Cell Death in Antiviral Immunity
Jason W. Upton et al.
MOLECULAR CELL (2014)
Plasma membrane translocation of trimerized MLKL protein is required for TNF-induced necroptosis
Zhenyu Cai et al.
NATURE CELL BIOLOGY (2014)
Direct activation of RIP3/MLKL-dependent necrosis by herpes simplex virus 1 (HSV-1) protein ICP6 triggers host antiviral defense
Xing Wang et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2014)
Programmed necrosis in microbial pathogenesis
Haripriya Sridharan et al.
TRENDS IN MICROBIOLOGY (2014)
Toll-like Receptor 3-mediated Necrosis via TRIF, RIP3, and MLKL
William J. Kaiser et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2013)
Viral modulation of programmed necrosis
William J. Kaiser et al.
CURRENT OPINION IN VIROLOGY (2013)
Mixed Lineage Kinase Domain-like Protein Mediates Necrosis Signaling Downstream of RIP3 Kinase
Liming Sun et al.
CELL (2012)
DAI/ZBP1/DLM-1 Complexes with RIP3 to Mediate Virus-Induced Programmed Necrosis that Is Targeted by Murine Cytomegalovirus vIRA
Jason W. Upton et al.
CELL HOST & MICROBE (2012)
The Amino Terminus of the Vaccinia Virus E3 Protein Is Necessary To Inhibit the Interferon Response
Stacy D. White et al.
JOURNAL OF VIROLOGY (2012)
Virus Inhibition of RIP3-Dependent Necrosis
Jason W. Upton et al.
CELL HOST & MICROBE (2010)
Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced Inflammation
YoungSik Cho et al.
CELL (2009)
Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-α
Sudan He et al.
CELL (2009)
DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-κB
Manuele Rebsamen et al.
EMBO REPORTS (2009)
RIP3, an Energy Metabolism Regulator That Switches TNF-Induced Cell Death from Apoptosis to Necrosis
Duan-Wu Zhang et al.
SCIENCE (2009)
Cytomegalovirus m45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1
Jason W. Upton et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
Receptor-Interacting Protein Homotypic Interaction Motif-Dependent Control of NF-κB Activation via the DNA-Dependent Activator of IFN Regulatory Factors
William J. Kaiser et al.
JOURNAL OF IMMUNOLOGY (2008)
TANK-binding kinase-1 delineates innate and adaptive immune responses to DNA vaccines
Ken J. Ishii et al.
NATURE (2008)
The N-terminal domain of the vaccinia virus E3L-protein is required for neurovirulence, but not induction of a protective immune response
T Brandt et al.
VIROLOGY (2005)
The solution structure of the N-terminal domain of E3L shows a tyrosine conformation that may explain its reduced affinity to Z-DNA in vitro
JD Kahmann et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2004)
Evidence that vaccinia virulence factor ER binds to Z-DNA in vivo:: Implications for development of a therapy for poxvirus infection
YG Kim et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2004)
Kinase RIP3 is dispensable for normal NF-KBs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and toll-like receptors 2 and 4
K Newton et al.
MOLECULAR AND CELLULAR BIOLOGY (2004)
A role for Z-DNA binding in vaccinia virus pathogenesis
YG Kim et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2003)
Both carboxy- and amino-terminal domains of the vaccinia virus interferon resistance gene, E3L, are required for pathogenesis in a mouse model
TA Brandt et al.
JOURNAL OF VIROLOGY (2001)
Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
N Holler et al.
NATURE IMMUNOLOGY (2000)
Induction of necrotic-like cell death by tumor necrosis factor alpha and caspase inhibitors: Novel mechanism for killing virus-infected cells
M Li et al.
JOURNAL OF VIROLOGY (2000)
Share Paper
