Journal
MOLECULAR & CELLULAR ONCOLOGY
Volume 6, Issue 6, Pages -Publisher
TAYLOR & FRANCIS INC
DOI: 10.1080/23723556.2019.1677140
Keywords
Retinoblastoma; cell cycle; senescence; nucleotide metabolism; mTORC1; reactive oxygen species; NF-kappa B; JNK1/3; SP1; AP-1
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Funding
- NIH/NCI grant [R00CA194309]
- Penn State Cancer Institute Postdoctoral Fellowship
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p16(INK4A) (hereafter called p16) is a faithful cellular ally in the fight against tumorigenesis. Although its canonical pathway through retinoblastoma (RB) is well delineated, RB-independent functions for p16 are beginning to emerge. Here we summarize non-canonical roles of p16, including our recent finding on its role in nucleotide metabolism.
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