Journal
JOURNAL OF CELL BIOLOGY
Volume 217, Issue 1, Pages 15-17Publisher
ROCKEFELLER UNIV PRESS
DOI: 10.1083/jcb.201711075
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Funding
- National Institutes of Health [GM61721]
- California Stem Cell Agency [RT307678]
- Deutsche Forschungsgemeinschaft [STE 2045/1-1]
- NATIONAL INSTITUTE OF GENERAL MEDICAL SCIENCES [R01GM061721] Funding Source: NIH RePORTER
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The formin-like protein INF2 is an important player in the polymerization of actin filaments. In this issue, Chakrabarti et al. (2018. J. Cell Biol. https://doi.org/10.1083/jcb.201709111) demonstrate that INF2 mediates actin polymerization at the endoplasmic reticulum (ER), resulting in increased ER-mitochondria contacts, calcium uptake by mitochondria, and mitochondrial division.
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