Related references
Note: Only part of the references are listed.An Entropy-Based Method for Identifying Mutual Exclusive Driver Genes in Cancer
Junrong Song et al.
IEEE-ACM TRANSACTIONS ON COMPUTATIONAL BIOLOGY AND BIOINFORMATICS (2020)
Identifying mutual exclusivity across cancer genomes: computational approaches to discover genetic interaction and reveal tumor vulnerability
Yulan Deng et al.
BRIEFINGS IN BIOINFORMATICS (2019)
MAP3K1 and MAP2K4 mutations are associated with sensitivity to MEK inhibitors in multiple cancer models
Zheng Xue et al.
CELL RESEARCH (2018)
Molecular landscape and targeted therapy of acute myeloid leukemia
Runxia Gu et al.
BIOMARKER RESEARCH (2018)
WeSME: uncovering mutual exclusivity of cancer drivers and beyond
Yoo-Ah Kim et al.
BIOINFORMATICS (2017)
The ribosomal protein gene RPL5 is a haploinsufficient tumor suppressor in multiple cancer types
Laura Fancello et al.
ONCOTARGET (2017)
BeWith: A Between-Within method to discover relationships between cancer modules via integrated analysis of mutual exclusivity, co-occurrence and functional interactions
Phuong Dao et al.
PLOS COMPUTATIONAL BIOLOGY (2017)
Functional roles of CTCF in breast cancer
Sumin Oh et al.
BMB REPORTS (2017)
MEGSA: A Powerful and Flexible Framework for Analyzing Mutual Exclusivity of Tumor Mutations
Xing Hua et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2016)
A weighted exact test for mutually exclusive mutations in cancer
Mark D. M. Leiserson et al.
BIOINFORMATICS (2016)
Functional Genomic Landscape of Human Breast Cancer Drivers, Vulnerabilities, and Resistance
Richard Marcotte et al.
CELL (2016)
SSA-ME Detection of cancer driver genes using mutual exclusivity by small subnetwork analysis
Sergio Pulido-Tamayo et al.
SCIENTIFIC REPORTS (2016)
A novel independence test for somatic alterations in cancer shows that biology drives mutual exclusivity but chance explains most co-occurrence
Sander Canisius et al.
GENOME BIOLOGY (2016)
MEMCover: integrated analysis of mutual exclusivity and functional network reveals dysregulated pathways across multiple cancer types
Yoo-Ah Kim et al.
BIOINFORMATICS (2015)
Systematic identification of cancer driving signaling pathways based on mutual exclusivity of genomic alterations
Ozgun Babur et al.
GENOME BIOLOGY (2015)
Abrogation of PIK3CA or PIK3R1 reduces proliferation, migration, and invasion in glioblastoma multiforme cells
Genevieve L. Weber et al.
Oncotarget (2015)
CoMEt: a statistical approach to identify combinations of mutually exclusive alterations in cancer
Mark D. M. Leiserson et al.
GENOME BIOLOGY (2015)
GATA3 mutations define a unique subtype of luminal-like breast cancer with improved survival
Yi-Zhou Jiang et al.
CANCER (2014)
How Many Etiological Subtypes of Breast Cancer: Two, Three, Four, Or More?
William F. Anderson et al.
JNCI-JOURNAL OF THE NATIONAL CANCER INSTITUTE (2014)
Discovery and saturation analysis of cancer genes across 21 tumour types
Michael S. Lawrence et al.
NATURE (2014)
Simultaneous Identification of Multiple Driver Pathways in Cancer
Mark D. M. Leiserson et al.
PLOS COMPUTATIONAL BIOLOGY (2013)
De novo discovery of mutated driver pathways in cancer
Fabio Vandin et al.
GENOME RESEARCH (2012)
Mutual exclusivity analysis identifies oncogenic network modules
Giovanni Ciriello et al.
GENOME RESEARCH (2012)
Prognostic impact, concurrent genetic mutations, and gene expression features of AML with CEBPA mutations in a cohort of 1182 cytogenetically normal AML patients: further evidence for CEBPA double mutant AML as a distinctive disease entity
Erdogan Taskesen et al.
BLOOD (2011)
Discovering functional modules by identifying recurrent and mutually exclusive mutational patterns in tumors
Christopher A. Miller et al.
BMC MEDICAL GENOMICS (2011)
Mutational Inactivation of STAG2 Causes Aneuploidy in Human Cancer
David A. Solomon et al.
SCIENCE (2011)
Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1
Roel G. W. Verhaak et al.
CANCER CELL (2010)
IDH1 Mutations as Molecular Signature and Predictive Factor of Secondary Glioblastomas
Sumihito Nobusawa et al.
CLINICAL CANCER RESEARCH (2009)
Implications of NRAS mutations in AML:: a study of 2502 patients
Ulrike Bacher et al.
BLOOD (2006)
PIK3CA mutations in glioblastoma multiforme
C Hartmann et al.
ACTA NEUROPATHOLOGICA (2005)
Share Paper
