Journal
TRENDS IN PARASITOLOGY
Volume 32, Issue 8, Pages 599-607Publisher
ELSEVIER SCI LTD
DOI: 10.1016/j.pt.2016.04.012
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Funding
- BBSRC [BB/L019035/1, BB/M012808/1, BB/N007492/1, BB/J004227/1]
- Bill & Melinda Gates Foundation
- GALVmed [UKAid (UK Government)]
- GALVmed [Bill & Melinda Gates Foundation]
- GALVmed
- Biotechnology and Biological Sciences Research Council [BB/L019035/1, BB/N007492/1, 1509513] Funding Source: researchfish
- BBSRC [BB/N007492/1, BB/L019035/1, BB/M012808/1] Funding Source: UKRI
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Animal African trypanosomiasis (AAT), caused by Trypanosome congolense and Trypanosoma vivax, remains one of the most important livestock diseases in sub-Saharan Africa, particularly affecting cattle. Despite this, our detailed knowledge largely stems from the human pathogen Trypanosoma brucei and mouse experimental models. In the postgenomic era, the genotypic and phenotypic differences between the AAT-relevant species of parasite or host and their model organism counterparts are increasingly apparent. Here, we outline the timeliness and advantages of increasing the research focus on both the clinically relevant parasite and host species, given that improved tools and resources for both have been developed in recent years. We propose that this shift of emphasis will improve our ability to efficiently develop tools to combat MT.
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