Journal
NATURE STRUCTURAL & MOLECULAR BIOLOGY
Volume 23, Issue 12, Pages 1070-1071Publisher
NATURE PUBLISHING GROUP
DOI: 10.1038/nsmb.3332
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Funding
- Intramural NIH HHS [Z99 AG999999] Funding Source: Medline
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Although oxidative stress has long been considered to be a major factor contributing to telomere shortening, recent work has established that oxidative stress and DNA damage are linked to telomere lengthening. Now, Opresko and colleagues resolve this apparent discrepancy by showing that differential modulation of telomerase activity depends on the origin of a common oxidative guanine lesion.
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