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STIM1 Gates TRPC Channels, but Not Orai1, by Electrostatic Interaction
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STIM2 is a feedback regulator that stabilizes basal cytosolic and endoplasmic reticulum Ca2+ levels
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Orai proteins interact with TRPC channels and confer responsiveness to store depletion
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Ca2+ refilling of the endoplasmic reticulum is largely preserved albeit reduced Ca2+ entry in endothelial cells
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Ca2+ influx induced by protease-activated receptor-1 activates a feed-forward mechanism of TRPC1 expression via nuclear factor-κB activation in endothelial cells
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CRACM1 is a plasma membrane protein essential for store-operated Ca2+ entry
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A mutation in Orai1 causes immune deficiency by abrogating CRAC channel function
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STIM1 carboxyl-terminus activates native SOC, I-crac and TRPC1 channels
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Sequential activation of store-operated currents in human gingival keratinocytes
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TRPC1 and TRPC5 form a novel cation channel in mammalian brain
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