4.6 Editorial Material

Does delayed EBV infection contribute to rising childhood cancers?

Journal

TRENDS IN IMMUNOLOGY
Volume 43, Issue 12, Pages 956-958

Publisher

CELL PRESS
DOI: 10.1016/j.it.2022.09.009

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Funding

  1. American Cancer Society Research Scholar Grant [RSG-19-035-01-LIB]
  2. Leukemia and Lymphoma Society [TRP-6595-20]
  3. Claudia Adams Barr Program for Innovative Cancer Research
  4. DGIST Start-up Fund Program of theMinistry of Science and ICT [2022010191]

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Childhood cancer is increasing in high-income countries, possibly due to reduced exposure to common infections in early life. The specific infection responsible for cancer protection has not been identified, but recent findings suggest that delayed Epstein-Barr virus (EBV) infection might contribute to the rise in childhood cancers.
Childhood cancer is on the rise in high-income countries. Epidemiological studies suggest that reduced exposure to common infections in early life is to blame. However, no specific infection responsible for protection against cancer has been identified, and the underlying mechanisms remain a matter of speculation. Recent findings that Epstein-Barr virus ( EBV) can induce antitumor immunity lead us to hypothesize that the delay in EBV infection in such countries might contribute to the increase in childhood cancers.

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