4.6 Article

The ryanodine receptor-calstabin interaction stabilizer S107 protects hippocampal neurons from GABAergic synaptic alterations induced by Abeta42 oligomers

Related references

Note: Only part of the references are listed.
Review Cell Biology

Ryanopathies and RyR2 dysfunctions: can we further decipher them using in vitro human disease models?

Yvonne Sleiman et al.

Summary: Regulation of intracellular calcium homeostasis is crucial for normal cell functions, with abnormal RyR2 function implicated in various diseases. Animal and human cell models have contributed valuable insights into RyR2 dysfunctions, offering potential for personalized therapeutics.

CELL DEATH & DISEASE (2021)

Review Neurosciences

Unsupervised excitation: GABAergic dysfunctions in Alzheimer's disease

Eleonora Ambrad Giovannetti et al.

BRAIN RESEARCH (2019)

Article Biochemistry & Molecular Biology

Ryanodine receptor dysfunction in human disorders

Alexander Kushnir et al.

BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH (2018)

Article Biochemistry & Molecular Biology

Amyloid β production is regulated by β2-adrenergic signaling-mediated post-translational modifications of the ryanodine receptor

Renaud Bussiere et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2017)

Review Clinical Neurology

Dysfunction of NMDA receptors in Alzheimer's disease

Yan Zhang et al.

NEUROLOGICAL SCIENCES (2016)

Review Physiology

Molecular Mechanisms for Synchronous, Asynchronous, and Spontaneous Neurotransmitter Release

Pascal S. Kaeser et al.

ANNUAL REVIEW OF PHYSIOLOGY, VOL 76 (2014)

Article Biochemistry & Molecular Biology

The role of ryanodine receptor type 3 in a mouse model of Alzheimer disease

Jie Liu et al.

CHANNELS (2014)

Review Pharmacology & Pharmacy

Calcium channelopathies and Alzheimer's disease: Insight into therapeutic success and failures

Shreaya Chakroborty et al.

EUROPEAN JOURNAL OF PHARMACOLOGY (2014)

Article Biochemistry & Molecular Biology

Reduction in neuronal L-type calcium channel activity in a double knock-in mouse model of Alzheimer's disease

Olivier Thibault et al.

BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2012)

Article Multidisciplinary Sciences

Stabilizing ER Ca2+ Channel Function as an Early Preventative Strategy for Alzheimer's Disease

Shreaya Chakroborty et al.

PLOS ONE (2012)

Article Biochemistry & Molecular Biology

Calcium Signalling and Alzheimer's Disease

Michael J. Berridge

NEUROCHEMICAL RESEARCH (2011)

Article Cell Biology

Ryanodine Receptors: Structure, Expression, Molecular Details, and Function in Calcium Release

Johanna T. Lanner et al.

COLD SPRING HARBOR PERSPECTIVES IN BIOLOGY (2010)

Article Multidisciplinary Sciences

Remodeling of ryanodine receptor complex causes leaky channels: A molecular mechanism for decreased exercise capacity

Andrew M. Bellinger et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)

Article Multidisciplinary Sciences

Clusters of hyperactive neurons near amyloid plaques in a mouse model of Alzheimer's disease

Marc Aurel Busche et al.

SCIENCE (2008)

Article Biochemistry & Molecular Biology

Amyloid-β-(1-42) increases ryanodine receptor-3 expression and function in neurons of TgCRND8 mice

Charlene Supnet et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2006)

Article Neurosciences

Regulation of NMDA receptor trafficking by amyloid-β

EM Snyder et al.

NATURE NEUROSCIENCE (2005)

Review Neurosciences

Vesicle pools and short-term synaptic depression: lessons from a large synapse

R Schneggenburger et al.

TRENDS IN NEUROSCIENCES (2002)

Article Neurosciences

Unveiling synaptic plasticity: a new graphical and analytical approach

JD Clements et al.

TRENDS IN NEUROSCIENCES (2000)