Related references
Note: Only part of the references are listed.Homeostatic p62 levels and inclusion body formation in CHCHD2 knockout mice
Shigeto Sato et al.
HUMAN MOLECULAR GENETICS (2021)
Loss of Parkin contributes to mitochondrial turnover and dopaminergic neuronal loss in aged mice
Sachiko Noda et al.
NEUROBIOLOGY OF DISEASE (2020)
Loss of FBXO7 results in a Parkinson's-like dopaminergic degeneration via an RPL23-MDM2-TP53 pathway
Simon R. W. Stott et al.
JOURNAL OF PATHOLOGY (2019)
Loss of autophagy in dopaminergic neurons causes Lewy pathology and motor dysfunction in aged mice
Shigeto Sato et al.
SCIENTIFIC REPORTS (2018)
Loss of FBXO7 (PARK15) results in reduced proteasome activity and models a parkinsonism-like phenotype in mice
Siv Vingill et al.
EMBO JOURNAL (2016)
The Parkinson's disease-linked proteins Fbxo7 and Parkin interact to mediate mitophagy
Victoria S. Burchell et al.
NATURE NEUROSCIENCE (2013)
PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy
Noriyuki Matsuda et al.
JOURNAL OF CELL BIOLOGY (2010)
FBXO7 mutations cause autosomal recessive, early-onset parkinsonian-pyramidal syndrome
A. Di Fonzo et al.
NEUROLOGY (2009)
Genome-wide linkage analysis of a Parkinsonian-pyramidal syndrome pedigree by 500 KSNP arrays
Seyedmehdi Shojaee et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2008)
Fission and selective fusion govern mitochondrial segregation and elimination by autophagy
Gilad Twig et al.
EMBO JOURNAL (2008)
Parkin is recruited selectively to impaired mitochondria and promotes their autophagy
Derek Narendra et al.
JOURNAL OF CELL BIOLOGY (2008)
Parkinson's disease brain mitochondrial complex I has oxidatively damaged subunits and is functionally impaired and misassembled
Paula M. Keeney et al.
JOURNAL OF NEUROSCIENCE (2006)