4.7 Article

Thermomechanical fatigue and fracture behaviours of welded joints at various temperatures

Journal

ENGINEERING FRACTURE MECHANICS
Volume 271, Issue -, Pages -

Publisher

PERGAMON-ELSEVIER SCIENCE LTD
DOI: 10.1016/j.engfracmech.2022.108644

Keywords

Welded joint; Thermomechanical fatigue; Temperature variation; Fracture behaviour

Categories

Funding

  1. National Natural Science Foundation of China [52005250]
  2. China Postdoctoral Science Foundation [2021M691559]
  3. Jiangsu Planned Projects for Postdoctoral Research Funds [2020Z320]

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This study investigates the relationship between temperature variation and thermomechanical fatigue (TMF) fracture behaviors. The results show that temperature changes lead to accelerated cyclic softening, reduced fatigue life, and dynamic strain aging (DSA), which are influenced by the temperature range and average temperature.
Temperature variation plays a crucial role in the safe operation of welded structures during longterm high-temperature service. The present work aimed to explore the relationship between temperature variation and thermomechanical fatigue (TMF) fracture behaviours in welded joints. To achieve this target, isothermal fatigue (IF) and TMF tests were performed on P92 steel welded joint at different average temperatures (500 degrees C, 550 degrees C, 600 degrees C, 650 degrees C) and different temperature ranges (0 degrees C, 100 degrees C, 200 degrees C). Results showed that the increases in the average temperature and temperature range induce accelerated cyclic softening, reduction in fatigue life, and more evident dynamic strain ageing (DSA). However, there is an obvious difference in the cyclic response between IF, in-phase TMF and out-of-phase TMF. The decline of friction stress is responsible for the reduced peak stress at a higher average temperature. The back stress plays a more critical role in the cyclic stress response with increasing the temperature range. Moreover, the different diffusion rates of solute atoms and formation rates of carbides at various temperatures are responsible for the pronounced difference in DSA activity. Notably, the fracture location changes with the variation of temperature, which is directly correlated to the evolution of fatigue life. The competitive mechanism between fatigue damage and creep damage is responsible for the shift of fracture location.

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