Journal
IMMUNITY
Volume 55, Issue 4, Pages 592-605Publisher
CELL PRESS
DOI: 10.1016/j.immuni.2022.03.016
Keywords
-
Categories
Funding
- Abby and Howard P. Milstein Program in Chemical Biology and Translational Medicine
- Randolph Hearst Trust
Ask authors/readers for more resources
Nonresolving inflammation contributes to various diseases, including COVID-19. Our understanding of inflammation is constantly evolving, viewing it as an interactive component of a homeostatic network with the endocrine and nervous systems. This review highlights emerging insights into inflammatory memory, inflammatory aging, inflammatory cell death, inflammatory DNA, inflammation-regulating cells and metabolites, approaches to resolving or modulating inflammation, and inflammatory inequity.
Nonresolving inflammation contributes to many diseases, including COVID-19 in its fatal and long forms. Our understanding of inflammation is rapidly evolving. Like the immune system of which it is a part, inflammation can now be seen as an interactive component of a homeostatic network with the endocrine and nervous systems. This review samples emerging insights regarding inflammatory memory, inflammatory aging, inflammatory cell death, inflammatory DNA, inflammation-regulating cells and metabolites, approaches to resolving or modulating inflammation, and inflammatory inequity.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available
Share Paper
