Related references
Note: Only part of the references are listed.The vitals of NETs
Chuyi Tan et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2021)
FDA-approved disulfiram inhibits pyroptosis by blocking gasdermin D pore formation
Jun Jacob Hu et al.
NATURE IMMUNOLOGY (2020)
Extracellular CIRP and TREM-1 axis promotes ICAM-1-Rho-mediated NETosis in sepsis
Atsushi Murao et al.
FASEB JOURNAL (2020)
Caspase-1 Engages Full-Length Gasdermin D through Two Distinct Interfaces That Mediate Caspase Recruitment and Substrate Cleavage
Zhonghua Liu et al.
IMMUNITY (2020)
Extracellular CIRP as an endogenous TREM-1 ligand to fuel inflammation in sepsis
Naomi-Liza Denning et al.
JCI INSIGHT (2020)
Pyroptosis engagement and bladder urothelial cell-derived exosomes recruit mast cells and induce barrier dysfunction of bladder urothelium after uropathogenic E. coli infection
Zonglong Wu et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2019)
Extracellular DNA traps in inflammation, injury and healing
Christoph Daniel et al.
NATURE REVIEWS NEPHROLOGY (2019)
The Caspase Inhibitor Z-VAD-FMK Alleviates Endotoxic Shock via Inducing Macrophages Necroptosis and Promoting MDSCs-Mediated Inhibition of Macrophages Activation
Xuehui Li et al.
FRONTIERS IN IMMUNOLOGY (2019)
Tissue-Specific Macrophage Responses to Remote Injury Impact the Outcome of Subsequent Local Immune Challenge
Friedrich Felix Hoyer et al.
IMMUNITY (2019)
Extracellular CIRP (eCIRP) and inflammation
Monowar Aziz et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2019)
Macrophage Extracellular Traps: A Scoping Review
Ryan S. Doster et al.
JOURNAL OF INNATE IMMUNITY (2018)
CIRP increases ICAM-1+ phenotype of neutrophils exhibiting elevated iNOS and NETs in sepsis
Yasumasa Ode et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2018)
Macrophage extracellular trap formation promoted by platelet activation is a key mediator of rhabdomyolysis-induced acute kidney injury
Koshu Okubo et al.
NATURE MEDICINE (2018)
Gasdermin D plays a vital role in the generation of neutrophil extracellular traps
Gabriel Sollberger et al.
SCIENCE IMMUNOLOGY (2018)
Pyroptosis: Gasdermin-Mediated Programmed Necrotic Cell Death
Jianjin Shi et al.
TRENDS IN BIOCHEMICAL SCIENCES (2017)
Pyroptosis and Apoptosis Pathways Engage in Bidirectional Crosstalk in Monocytes and Macrophages
Cornelius Y. Taabazuing et al.
CELL CHEMICAL BIOLOGY (2017)
Cold-inducible RNA-binding protein causes endothelial dysfunction via activation of Nlrp3 inflammasome
Weng-Lang Yang et al.
SCIENTIFIC REPORTS (2016)
Paradoxical Roles of the neutrophil in Sepsis: Protective and Deleterious
Fabiane Sonego et al.
FRONTIERS IN IMMUNOLOGY (2016)
Extracellular traps and macrophages: new roles for the versatile phagocyte
Devin M. Boe et al.
JOURNAL OF LEUKOCYTE BIOLOGY (2015)
Caspase-11 cleaves gasdermin D for non-canonical inflammasome signalling
Nobuhiko Kayagaki et al.
NATURE (2015)
Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death
Jianjin Shi et al.
NATURE (2015)
Caspase-11 Controls Interleukin-1β Release through Degradation of TRPC1
Benedicte F. Py et al.
CELL REPORTS (2014)
Cold-inducible RNA-binding protein (CIRP) triggers inflammatory responses in hemorrhagic shock and sepsis
Xiaoling Qiang et al.
NATURE MEDICINE (2013)
Intravascular Neutrophil Extracellular Traps Capture Bacteria from the Bloodstream during Sepsis
Braedon McDonald et al.
CELL HOST & MICROBE (2012)
Mannheimia haemolytica and Its Leukotoxin Cause Macrophage Extracellular Trap Formation by Bovine Macrophages
Nicole A. Aulik et al.
INFECTION AND IMMUNITY (2012)
Fiji: an open-source platform for biological-image analysis
Johannes Schindelin et al.
NATURE METHODS (2012)
PAD4 mediated histone hypercitrullination induces heterochromatin decondensation and chromatin unfolding to form neutrophil extracellular trap-like structures
Marc Leshner et al.
FRONTIERS IN IMMUNOLOGY (2012)
Statins Enhance Formation of Phagocyte Extracellular Traps
Ohn A. Chow et al.
CELL HOST & MICROBE (2010)
Broad-spectrum caspase inhibitors: from myth to reality?
D. Chauvier et al.
CELL DEATH AND DIFFERENTIATION (2007)
Uncoupling cell shrinkage from apoptosis reveals that Na+ influx is required for volume loss during programmed cell death
CD Bortner et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)