Journal
TRENDS IN ENDOCRINOLOGY AND METABOLISM
Volume 33, Issue 1, Pages 1-3Publisher
CELL PRESS
DOI: 10.1016/j.tem.2021.11.001
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Funding
- Societe Francophone du Diabete
- Fondation Genavie
- INSERM [ANR-16-CE92-0032-02, ANR-16-CE14-0007-01]
- Idex Universite de Paris
- Fondation pour la Recherche Medicale [EQU202003010767]
- Universited'Angers
- Agence Nationale de la Recherche (ANR) [ANR-16-CE14-0007, ANR-16-CE92-0032] Funding Source: Agence Nationale de la Recherche (ANR)
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Research shows that nutrient excess leads to mitochondrial dysfunction, contributing to obesity-related complications, while adipocyte-derived extracellular vesicles play a protective role against cardiac oxidative stress.
Nutrient excess induces mitochondrial dysfunction, which participates in obesity-related complications. Obesity also associates with high cardiac oxidative stress, which contributes to myocardial dysfunction. Crewe et al. recently evidenced the pivotal role of adipocyte-derived extracellular vesicles (EVs) in cardiac oxidative stress responses and revealed their unexpected protective effect against ischemia/reperfusion injury.
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