Related references
Note: Only part of the references are listed.Silencing TAK1 reduces MAPKs-MMP2/9 expression to reduce inflammation-driven neurohistological disruption post spinal cord injury
Shuai Jiang et al.
CELL DEATH DISCOVERY (2021)
Structural basis for specific recognition of K6-linked polyubiquitin chains by the TAB2 NZF domain
Yanjun Li et al.
BIOPHYSICAL JOURNAL (2021)
The SUMOylation of TAB2 mediated by TRIM60 inhibits MAPK/NF-κB activation and the innate immune response
Zhiwen Gu et al.
CELLULAR & MOLECULAR IMMUNOLOGY (2021)
Insights into the molecular pathogenesis of cardiospondylocarpofacial syndrome: MAP3K7 c.737-7A > G variant alters the TGFβ-mediated α-SMA cytoskeleton assembly and autophagy
Lucia Micale et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2020)
A novel TAB2 nonsense mutation (p.S149X) causing autosomal dominant congenital heart defects: a case report of a Chinese family
Jia Chen et al.
BMC CARDIOVASCULAR DISORDERS (2020)
A single nucleotide deletion resulting in a frameshift in exon 4 of TAB2 is associated with a polyvalular syndrome
Eduard Permanyer et al.
EUROPEAN JOURNAL OF MEDICAL GENETICS (2020)
Next-generation sequencing in a series of 80 fetuses with complex cardiac malformations and/or heterotaxy
Hui Liu et al.
HUMAN MUTATION (2020)
TAB2 c.1398dup variant leads to haploinsufficiency and impairs extracellular matrix homeostasis
Silvia Morlino et al.
HUMAN MUTATION (2019)
TRIM38 regulates NF-kappa B activation through TAB2 degradation in osteoclast and osteoblast differentiation
Kabsun Kim et al.
BONE (2018)
A novel MAP3K7 splice mutation causes cardiospondylocarpofacial syndrome with features of hereditary connective tissue disorder
Silvia Morlino et al.
EUROPEAN JOURNAL OF HUMAN GENETICS (2018)
Effect of TAK1 on osteogenic differentiation of mesenchymal stem cells by regulating BMP-2 via Wnt/βcatenin and MAPK pathway
Hongpeng Yang et al.
ORGANOGENESIS (2018)
Protein molecular modeling techniques investigating novel TAB2 variant R347X causing cardiomyopathy and congenital heart defects in multigenerational family
Thomas R. Caulfield et al.
MOLECULAR GENETICS & GENOMIC MEDICINE (2018)
A recognizable systemic connective tissue disorder with polyvalvular heart dystrophy and dysmorphism associated with TAB2 mutations
M. Ritelli et al.
CLINICAL GENETICS (2018)
Post-Translational Modifications of the TAK1-TAB Complex
Yusuke Hirata et al.
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES (2017)
6q25.1 (TAB2) microdeletion syndrome: Congenital heart defects and cardiomyopathy
Andrew Cheng et al.
AMERICAN JOURNAL OF MEDICAL GENETICS PART A (2017)
Autosomal dominant frontometaphyseal dysplasia: Delineation of the clinical phenotype
Emma M. Wade et al.
AMERICAN JOURNAL OF MEDICAL GENETICS PART A (2017)
Mutations in MAP3K7 that Alter the Activity of the TAK1 Signaling Complex Cause Frontometaphyseal Dysplasia
Emma M. Wade et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2016)
Whole Exome Sequencing, Familial Genomic Triangulation, and Systems Biology Converge to Identify a Novel Nonsense Mutation in TAB2-encoded TGF-beta Activated Kinase 1 in a Child with Polyvalvular Syndrome
Jaeger P. Ackerman et al.
CONGENITAL HEART DISEASE (2016)
Biomineralization Guided by Paper Templates
Gulden Camci-Unal et al.
SCIENTIFIC REPORTS (2016)
Familial TAB2 microdeletion and congenital heart defects including unusual valve dysplasia and tetralogy of fallot
Karin Weiss et al.
AMERICAN JOURNAL OF MEDICAL GENETICS PART A (2015)
A De Novo 0.63 Mb 6q25.1 Deletion Associated with Growth Failure, Congenital Heart Defect, Underdeveloped Cerebellar Vermis, Abnormal Cutaneous Elasticity and Joint Laxity
Vincenzo Salpietro et al.
AMERICAN JOURNAL OF MEDICAL GENETICS PART A (2015)
Reciprocal Inhibition between the Transforming Growth Factor-β-activated Kinase 1 (TAK1) and Apoptosis Signal-regulating Kinase 1 (ASK1) Mitogen-activated Protein Kinase Kinase Kinases and Its Suppression by TAK1-binding Protein 2 (TAB2), an Adapter Protein for TAK1
So Yong Kim et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2012)
Haploinsufficiency of TAB2 Causes Congenital Heart Defects in Humans
Bernard Thienpont et al.
AMERICAN JOURNAL OF HUMAN GENETICS (2010)
A scaling normalization method for differential expression analysis of RNA-seq data
Mark D. Robinson et al.
GENOME BIOLOGY (2010)
Structural basis for specific recognition of Lys 63-linked polyubiquitin chains by NZF domains of TAB2 and TAB3
Yusuke Sato et al.
EMBO JOURNAL (2009)
The type I TGF-β receptor engages TRAF6 to activate TAK1 in a receptor kinase-independent manner
Alessandro Sorrentino et al.
NATURE CELL BIOLOGY (2008)
TRAF6 Autoubiquitination-Independent Activation of the NFκB and MAPK Pathways in Response to IL-1 and RANKL
Matthew C. Walsh et al.
PLOS ONE (2008)
Disabled-2 (Dab2) mediates transforming growth factor β (TGFβ)-stimulated fibronectin synthesis through TGFβ-activated kinase 1 and activation of the JNK pathway
BA Hocevar et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
TAB2 and TAB3 activate the NF-κB pathway through binding to polyubiquitin chains
A Kanayama et al.
MOLECULAR CELL (2004)
TAK1 is a ubiquitin-dependent kinase of MKK and IKK
C Wang et al.
NATURE (2001)
Stimulation of pro-α1(I) collagen by TGF-β1 in mesangial cells:: role of the p38 MAPK pathway
BY Chin et al.
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2001)
TAB2, a novel adaptor protein, mediates activation of TAK1 MAPKKK by linking TAK1 to TRAF6 in the IL-1 signal transduction pathway
G Takaesu et al.
MOLECULAR CELL (2000)
Share Paper
