4.8 Article

Growth of Mycobacterium tuberculosis at acidic pH depends on lipid assimilation and is accompanied by reduced GAPDH activity

Related references

Note: Only part of the references are listed.
Article Biochemistry & Molecular Biology

Allosteric regulation of pyruvate kinase from Mycobacterium tuberculosis by metabolites

Jan Snasel et al.

BIOCHIMICA ET BIOPHYSICA ACTA-PROTEINS AND PROTEOMICS (2019)

Article Multidisciplinary Sciences

Subcellular antibiotic visualization reveals a dynamic drug reservoir in infected macrophages

Daniel J. Greenwood et al.

SCIENCE (2019)

Article Cell Biology

Targeting redox heterogeneity to counteract drug tolerance in replicating Mycobacterium tuberculosis

Richa Mishra et al.

SCIENCE TRANSLATIONAL MEDICINE (2019)

Article Multidisciplinary Sciences

Genetic and metabolic regulation of Mycobacterium tuberculosis acid growth arrest

Jacob J. Baker et al.

SCIENTIFIC REPORTS (2018)

Article Multidisciplinary Sciences

Glyoxylate detoxification is an essential function of malate synthase required for carbon assimilation in Mycobacterium tuberculosis

Susan Puckett et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2017)

Article Microbiology

Lung Tissue Concentrations of Pyrazinamide among Patients with Drug-Resistant Pulmonary Tuberculosis

Russell R. Kempker et al.

ANTIMICROBIAL AGENTS AND CHEMOTHERAPY (2017)

Article Biochemistry & Molecular Biology

Mycobacterium tuberculosis protease MarP activates a peptidoglycan hydrolase during acid stress

Helene Botella et al.

EMBO JOURNAL (2017)

Review Biochemistry & Molecular Biology

Cytosolic thiol switches regulating basic cellular functions: GAPDH as an information hub?

Thomas Hildebrandt et al.

BIOLOGICAL CHEMISTRY (2015)

Article Biochemistry & Molecular Biology

Slow growth of Mycobacterium tuberculosis at acidic pH is regulated by phoPR and host-associated carbon sources

Jacob J. Baker et al.

MOLECULAR MICROBIOLOGY (2014)

Article Biochemistry & Molecular Biology

Intracellular Mycobacterium tuberculosis Exploits Host-derived Fatty Acids to Limit Metabolic Stress

Wonsik Lee et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2013)

Article Biochemistry & Molecular Biology

Metabolomics of Mycobacterium tuberculosis Reveals Compartmentalized Co-Catabolism of Carbon Substrates

Luiz Pedro S. de Carvalho et al.

CHEMISTRY & BIOLOGY (2010)

Article Medicine, Research & Experimental

Caseation of human tuberculosis granulomas correlates with elevated host lipid metabolism

Mi-Jeong Kim et al.

EMBO MOLECULAR MEDICINE (2010)

Article Biotechnology & Applied Microbiology

Acid tolerance in Salmonella typhimurium induced by culturing in the presence of organic acids at different growth temperatures

Avelino Alvarez-Ordonez et al.

FOOD MICROBIOLOGY (2010)

Article Multidisciplinary Sciences

Gluconeogenic carbon flow of tricarboxylic acid cycle intermediates is critical for Mycobacterium tuberculosis to establish and maintain infection

Joeli Marrero et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2010)

Article Biochemistry & Molecular Biology

A membrane protein preserves intrabacterial pH in intraphagosomal Mycobacterium tuberculosis

Omar H. Vandal et al.

NATURE MEDICINE (2008)

Article Multidisciplinary Sciences

Mycobacterial persistance requires the utilization of host cholesterol

Amit K. Pandey et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)

Article Biochemistry & Molecular Biology

Mycobacterium tuberculosis isocitrate lyases 1 and 2 are jointly required for in vivo growth and virulence

EJ Muñoz-Elías et al.

NATURE MEDICINE (2005)

Article Multidisciplinary Sciences

Immune control of tuberculosis by IFN-γ-inducible LRG-47

JD MacMicking et al.

SCIENCE (2003)

Article Biotechnology & Applied Microbiology

Acid tolerance of Shigella sonnei and Shigella flexneri

CF Bagamboula et al.

JOURNAL OF APPLIED MICROBIOLOGY (2002)