Related references
Note: Only part of the references are listed.Pancancer survival analysis of cancer hallmark genes
Adam Nagy et al.
SCIENTIFIC REPORTS (2021)
Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases
Elena Perenthaler et al.
ACTA NEUROPATHOLOGICA (2020)
Site-Specific Glycosylation Quantitation of 50 Serum Glycoproteins Enhanced by Predictive Glycopeptidomics for Improved Disease Biomarker Discovery
Qiongyu Li et al.
ANALYTICAL CHEMISTRY (2019)
Identification of UGP2 as a progression marker that promotes cell growth and motility in human glioma
Chun Zeng et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2019)
UDP-glucose accelerates SNAI1 mRNA decay and impairs lung cancer metastasis
Xiongjun Wang et al.
NATURE (2019)
Identification of potential sialic acid binding proteins on cell membranes by proximity chemical labeling
Qiongyu Li et al.
CHEMICAL SCIENCE (2019)
Unveiling the metabolic fate of monosaccharides in cell membranes with glycomic and glycoproteomic analyses
Gege Xu et al.
CHEMICAL SCIENCE (2019)
Multiomics Integration Reveals the Landscape of Prometastasis Metabolism in Hepatocellular Carcinoma
Yongmei Li et al.
MOLECULAR & CELLULAR PROTEOMICS (2018)
Expression of UGP2 and CFL1 expression levels in benign and malignant pancreatic lesions and their clinicopathological significance
Lingxiang Wang et al.
WORLD JOURNAL OF SURGICAL ONCOLOGY (2018)
Membrane glycomics reveal heterogeneity and quantitative distribution of cell surface sialylation
Diane Dayoung Park et al.
CHEMICAL SCIENCE (2018)
Identifying mouse developmental essential genes using machine learning
David Tian et al.
DISEASE MODELS & MECHANISMS (2018)
Computational correction of copy number effect improves specificity of CRISPR-Cas9 essentiality screens in cancer cells
Robin M. Meyers et al.
NATURE GENETICS (2017)
Overexpression of Yes Associated Protein 1, an Independent Prognostic Marker in Patients With Pancreatic Ductal Adenocarcinoma, Correlated With Liver Metastasis and Poor Prognosis
Maria Teresa Salcedo Allende et al.
PANCREAS (2017)
Drug resistance in pancreatic cancer: Impact of altered energy metabolism
Cristoforo Grasso et al.
CRITICAL REVIEWS IN ONCOLOGY HEMATOLOGY (2017)
SHP2 and UGP2 are Biomarkers for Progression and Poor Prognosis of Gallbladder Cancer
Qunwei Wang et al.
CANCER INVESTIGATION (2016)
Genetics and biology of pancreatic ductal adenocarcinoma
Haoqiang Ying et al.
GENES & DEVELOPMENT (2016)
Inactivation of hypoxia-induced YAP by statins overcomes hypoxic resistance tosorafenib in hepatocellular carcinoma cells
Tian-yi Zhou et al.
SCIENTIFIC REPORTS (2016)
Human Pancreatic Cancer Tumors Are Nutrient Poor and Tumor Cells Actively Scavenge Extracellular Protein
Jurre J. Kamphorst et al.
CANCER RESEARCH (2015)
Characteristic Changes in Cell Surface Glycosylation Accompany Intestinal Epithelial Cell (IEC) Differentiation: High Mannose Structures Dominate the Cell Surface Glycome of Undifferentiated Enterocytes
Dayoung Park et al.
MOLECULAR & CELLULAR PROTEOMICS (2015)
N-Glycosylation as determinant of epidermal growth factor receptor conformation in membranes
Karol Kaszuba et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2015)
Targeting cancer cell metabolism in pancreatic adenocarcinoma
Romain Cohen et al.
ONCOTARGET (2015)
A Quaternary Mechanism Enables the Complex Biological Functions of Octameric Human UDP-glucose Pyrophosphorylase, a Key Enzyme in Cell Metabolism
Jana Indra Fuehring et al.
SCIENTIFIC REPORTS (2015)
YAP1 Exerts Its Transcriptional Control via TEAD-Mediated Activation of Enhancers
Claudia Stein et al.
PLOS GENETICS (2015)
Glycogen metabolism in cancer
Christos E. Zois et al.
BIOCHEMICAL PHARMACOLOGY (2014)
Yap1 Activation Enables Bypass of Oncogenic Kras Addiction in Pancreatic Cancer
Avnish Kapoor et al.
CELL (2014)
KRAS and YAP1 Converge to Regulate EMT and Tumor Survival
Diane D. Shao et al.
CELL (2014)
Downstream of Mutant KRAS, the Transcription Regulator YAP Is Essential for Neoplastic Progression to Pancreatic Ductal Adenocarcinoma
Weiying Zhang et al.
SCIENCE SIGNALING (2014)
Macropinocytosis of protein is an amino acid supply route in Ras-transformed cells
Cosimo Commisso et al.
NATURE (2013)
Oncogenic Kras Maintains Pancreatic Tumors through Regulation of Anabolic Glucose Metabolism
Haoqiang Ying et al.
CELL (2012)
Glucose Utilization via Glycogen Phosphorylase Sustains Proliferation and Prevents Premature Senescence in Cancer Cells
Elena Favaro et al.
CELL METABOLISM (2012)
Extensive Determination of Glycan Heterogeneity Reveals an Unusual Abundance of High Mannose Glycans in Enriched Plasma Membranes of Human Embryonic Stem Cells
Hyun Joo An et al.
MOLECULAR & CELLULAR PROTEOMICS (2012)
Glycogen synthesis is induced in hypoxia by the hypoxia-inducible factor and promotes cancer cell survival
Joffrey Pelletier et al.
FRONTIERS IN ONCOLOGY (2012)
Gene expression profiling in the leukemic stem cell-enriched CD34+ fraction identifies target genes that predict prognosis in normal karyotype AML
H. J. M. de Jonge et al.
LEUKEMIA (2011)
Hypoxia Promotes Glycogen Accumulation through Hypoxia Inducible Factor (HIF)-Mediated Induction of Glycogen Synthase 1
Nuria Pescador et al.
PLOS ONE (2010)
TEAD mediates YAP-dependent gene induction and growth control
Bin Zhao et al.
GENES & DEVELOPMENT (2008)
Inactivation of YAP oncoprotein by the Hippo pathway is involved in cell contact inhibition and tissue growth control
Bin Zhao et al.
GENES & DEVELOPMENT (2007)
Metabolic sensitivity of pancreatic tumour cell apoptosis to glycogen phosphorylase inhibitor treatment
WNP Lee et al.
BRITISH JOURNAL OF CANCER (2004)
A point mutation in the UDP-glucose pyrophosphorylase gene results in decreases of UDP-glucose and inactivation of glycogen synthase
JC Higuita et al.
BIOCHEMICAL JOURNAL (2003)
TEAD/TEF transcription factors utilize the activation domain of YAP65, a Src/Yes-associated protein localized in the cytoplasm
A Vassilev et al.
GENES & DEVELOPMENT (2001)