Journal
ENVIRONMENTAL SCIENCE AND POLLUTION RESEARCH
Volume 28, Issue 39, Pages 54429-54447Publisher
SPRINGER HEIDELBERG
DOI: 10.1007/s11356-021-15556-0
Keywords
PM2 5; PM10; Nitrogen dioxide (NO2); Ozone (O-3); Wildfires; Smoking; Covid 19; SARS Cov 2
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Outdoor levels of PM2.5, CO, and other pollutants are linked to higher rates of Covid 19 morbidity and mortality; smoking, wildfires, and exposure to incense, pesticides, heavy metals are also associated with increased risk of Covid 19 cases and deaths; the exact mechanisms of how air pollutants increase Covid 19 infections are not fully understood, but are likely related to oxidation and inflammation in the lungs and other tissues.
Numerous studies have linked outdoor levels of PM2.5, PM10, NO2, O-3, SO2, and other air pollutants to significantly higher rates of Covid 19 morbidity and mortality, although the rate in which specific concentrations of pollutants increase Covid 19 morbidity and mortality varies widely by specific country and study. As little as a 1-mu g/m(3) increase in outdoor PM2.5 is estimated to increase rates of Covid 19 by as much as 0.22 to 8%. Two California studies have strongly linked heavy wildfire burning periods with significantly higher outdoor levels of PM2.5 and CO as well as significantly higher rates of Covid 19 cases and deaths. Active smoking has also been strongly linked significantly increased risk of Covid 19 severity and death. Other exposures possibly related to greater risk of Covid 19 morbidity and mortality include incense, pesticides, heavy metals, dust/sand, toxic waste sites, and volcanic emissions. The exact mechanisms in which air pollutants increase Covid 19 infections are not fully understood, but are probably related to pollutant-related oxidation and inflammation of the lungs and other tissues and to the pollutant-driven alternation of the angiotensin-converting enzyme 2 in respiratory and other cells.
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