Related references
Note: Only part of the references are listed.PPARγ and RhoBTB1 in hypertension
Shi Fang et al.
CURRENT OPINION IN NEPHROLOGY AND HYPERTENSION (2020)
Preeclampsia and Kidney Disease: Deciphering Cause and Effect
Andrea Kattah
CURRENT HYPERTENSION REPORTS (2020)
RhoBTB1 protects against hypertension and arterial stiffness by restraining phosphodiesterase 5 activity
Masashi Mukohda et al.
JOURNAL OF CLINICAL INVESTIGATION (2019)
Molecular determinants of microvascular dysfunction in hypertensive pregnancy and preeclampsia
Wentao Yu et al.
MICROCIRCULATION (2019)
Conditional deletion of smooth muscle Cullin-3 causes severe progressive hypertension
Larry N. Agbor et al.
JCI INSIGHT (2019)
Dual gain and loss of cullin 3 function mediates familial hyperkalemic hypertension
Ryan J. Cornelius et al.
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2018)
With no lysine kinase 4 modulates sodium potassium 2 chloride cotransporter activity in vivo
Andrew S. Terker et al.
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2018)
WNK1 is required for proliferation induced by hypotonic challenge in rat vascular smooth muscle cells
Ya-juan Zhang et al.
ACTA PHARMACOLOGICA SINICA (2018)
The effects of sildenafil citrate on urinary podocin and nephrin mRNA expression in an L-NAME model of pre-eclampsia
Sooraj Baijnath et al.
MOLECULAR AND CELLULAR BIOCHEMISTRY (2017)
Low Soluble Fms-Like Tyrosine Kinase-1, Endoglin, and Endothelin-1 Levels in Women With Confirmed or Suspected Preeclampsia Using Proton Pump Inhibitors
Langeza Saleh et al.
HYPERTENSION (2017)
Proton Pump Inhibitors Decrease Soluble fms-Like Tyrosine Kinase-1 and Soluble Endoglin Secretion, Decrease Hypertension, and Rescue Endothelial Dysfunction
Kenji Onda et al.
HYPERTENSION (2017)
Constitutively Active SPAK Causes Hyperkalemia by Activating NCC and Remodeling Distal Tubules
P. Richard Grimm et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2017)
Urine exosomes from healthy and hypertensive pregnancies display elevated level of α-subunit and cleaved α- and γ-subunits of the epithelial sodium channel-ENaC
Maria R. Nielsen et al.
PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY (2017)
Mutant Cullin 3 causes familial hyperkalemic hypertension via dominant effects
Mohammed Z. Ferdaus et al.
JCI INSIGHT (2017)
Involvement of selective autophagy mediated by p62/SQSTM1 in KLHL3-dependent WNK4 degradation
Yutaro Mori et al.
BIOCHEMICAL JOURNAL (2015)
Bioactive factors in uteroplacental and systemic circulation link placental ischemia to generalized vascular dysfunction in hypertensive pregnancy and preeclampsia
Dania A. Shah et al.
BIOCHEMICAL PHARMACOLOGY (2015)
Potassium Modulates Electrolyte Balance and Blood Pressure through Effects on Distal Cell Voltage and Chloride
Andrew S. Terker et al.
CELL METABOLISM (2015)
Genetic, immune and vasoactive factors in the vascular dysfunction associated with hypertension in pregnancy
Sajjadh M. J. Ali et al.
EXPERT OPINION ON THERAPEUTIC TARGETS (2015)
Hypoxia-Independent Upregulation of Placental Hypoxia Inducible Factor-1α Gene Expression Contributes to the Pathogenesis of Preeclampsia
Takayuki Iriyama et al.
HYPERTENSION (2015)
Kelch-Like Protein 2 Mediates Angiotensin II-With No Lysine 3 Signaling in the Regulation of Vascular Tonus
Moko Zeniya et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2015)
New insights into the function of Cullin 3 in trophoblast invasion and migration
Qian Zhang et al.
REPRODUCTION (2015)
Hyperkalemic hypertension-associated cullin 3 promotes WNK signaling by degrading KLHL3
James A. McCormick et al.
JOURNAL OF CLINICAL INVESTIGATION (2014)
Vascular Dysfunction in Preeclampsia
Lesley J. Brennan et al.
MICROCIRCULATION (2014)
Global causes of maternal death: a WHO systematic analysis
Lale Say et al.
LANCET GLOBAL HEALTH (2014)
Innate immune system and preeclampsia
Alejandra Perez-Sepulveda et al.
FRONTIERS IN IMMUNOLOGY (2014)
Change of Genes in Calcium Transport Channels Caused by Hypoxic Stress in the Placenta, Duodenum, and Kidney of Pregnant Rats
Hyun Yang et al.
BIOLOGY OF REPRODUCTION (2013)
Kelch-like 3 and Cullin 3 regulate electrolyte homeostasis via ubiquitination and degradation of WNK4
Shigeru Shibata et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2013)
Natriuria and calciuria levels in preeclampsia: a cross-sectional study
Rose Gasnier et al.
SAO PAULO MEDICAL JOURNAL (2013)
Cullin-3 Regulates Vascular Smooth Muscle Function and Arterial Blood Pressure via PPARγ and RhoA/Rho-Kinase
Christopher J. Pelham et al.
CELL METABOLISM (2012)
Altered expression of COP9 signalosome proteins in preeclampsia
Sevil Cayli et al.
GYNECOLOGICAL ENDOCRINOLOGY (2012)
Mutations in kelch-like 3 and cullin 3 cause hypertension and electrolyte abnormalities
Lynn M. Boyden et al.
NATURE (2012)
Circulating activators of peroxisome proliferator-activated receptors are reduced in preeclamptic pregnancy
LL Waite et al.
JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM (2005)
Jab1 interacts directly with HIF-1α and regulates its stability
MK Bae et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Reduced uterine perfusion pressure during pregnancy in the rat is associated with increases in arterial pressure and changes in renal nitric oxide
BT Alexander et al.
HYPERTENSION (2001)
Share Paper
