Related references
Note: Only part of the references are listed.Cardiac Pacemaker Activity and Aging
Colin H. Peters et al.
ANNUAL REVIEW OF PHYSIOLOGY, VOL 82 (2020)
Mechanism of adrenergic CaV1.2 stimulation revealed by proximity proteomics
Guoxia Liu et al.
NATURE (2020)
Neurohumoral Control of Sinoatrial Node Activity and Heart Rate: Insight From Experimental Models and Findings From Humans
Eilidh A. MacDonald et al.
FRONTIERS IN PHYSIOLOGY (2020)
Cells of the adult human heart
Monika Litvinukova et al.
NATURE (2020)
Myocardial-restricted ablation of the GTPase RAD results in a pro-adaptive heart response in mice
Brooke M. Ahern et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2019)
Quantitative proteomics and single-nucleus transcriptomics of the sinus node elucidates the foundation of cardiac pacemaking
Nora Linscheid et al.
NATURE COMMUNICATIONS (2019)
Cardiac CaV1.2 channels require β subunits for β-adrenergic-mediated modulation but not trafficking
Lin Yang et al.
JOURNAL OF CLINICAL INVESTIGATION (2019)
Positive Feedback Mechanisms among Local Ca Releases, NCX, and ICaL Ignite Pacemaker Action Potentials
Alexey E. Lyashkov et al.
BIOPHYSICAL JOURNAL (2018)
Elevated plasma catecholamines functionally compensate for the reduced myogenic tone in smooth muscle STIM1 knockout mice but with deleterious cardiac effects
Prahalathan Pichavaram et al.
CARDIOVASCULAR RESEARCH (2018)
Heterogeneity of calcium clock functions in dormant, dysrhythmically and rhythmically firing single pacemaker cells isolated from SA node
Mary S. Kim et al.
CELL CALCIUM (2018)
2017 ACC/AHA/HFSA Focused Update of the 2013 ACCF/AHA Guideline for the Management of Heart Failure
Clyde W. Yancy et al.
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY (2017)
Redundant and diverse intranodal pacemakers and conduction pathways protect the human sinoatrial node from failure
Ning Li et al.
SCIENCE TRANSLATIONAL MEDICINE (2017)
Ivabradine in Heart Failure The Representativeness of SHIFT (Systolic Heart Failure Treatment With the IF Inhibitor Ivabradine Trial) in a Broad Population of Patients With Chronic Heart Failure
Debraj Das et al.
CIRCULATION-HEART FAILURE (2017)
Rad GTPase is essential for the regulation of bone density and bone marrow adipose tissue in mice
Catherine N. Withers et al.
BONE (2017)
L-type Cav1.3 channels regulate ryanodine receptor-dependent Ca2+ release during sino-atrial node pacemaker activity
Angelo Giovanni Torrente et al.
CARDIOVASCULAR RESEARCH (2016)
Rad-deletion Phenocopies Tonic Sympathetic Stimulation of the Heart
Bryana M. Levitan et al.
JOURNAL OF CARDIOVASCULAR TRANSLATIONAL RESEARCH (2016)
Loss of Rad-GTPase produces a novel adaptive cardiac phenotype resistant to systolic decline with aging
Janet R. Manning et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2015)
Signaling effectors underlying pathologic growth and remodeling of the heart
Jop H. van Berlo et al.
JOURNAL OF CLINICAL INVESTIGATION (2013)
Depressed pacemaker activity of sinoatrial node myocytes contributes to the age-dependent decline in maximum heart rate
Eric D. Larson et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2013)
Rad GTPase Deletion Increases L-type Calcium Channel Current Leading to Increased Cardiac Contraction
Janet R. Manning et al.
JOURNAL OF THE AMERICAN HEART ASSOCIATION (2013)
Deletion of the C-terminal Phosphorylation Sites in the Cardiac β-Subunit Does Not Affect the Basic β-Adrenergic Response of the Heart and the Cav1.2 Channel
Julia Brandmayr et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2012)
RNAscope A Novel in Situ RNA Analysis Platform for Formalin-Fixed, Paraffin-Embedded Tissues
Fay Wang et al.
JOURNAL OF MOLECULAR DIAGNOSTICS (2012)
Distinct localization and modulation of Cav1.2 and Cav1.3 L-type Ca2+ channels in mouse sinoatrial node
Carl J. Christel et al.
JOURNAL OF PHYSIOLOGY-LONDON (2012)
Rem GTPase interacts with the proximal CaV1.2 C-terminus and modulates calcium-dependent channel inactivation
Chunyan Pang et al.
CHANNELS (2010)
Rad As a Novel Regulator of Excitation-Contraction Coupling and β-Adrenergic Signaling in Heart
Gang Wang et al.
CIRCULATION RESEARCH (2010)
Increased catecholamine secretion contributes to hypertension in TRPM4-deficient mice
Ilka Mathar et al.
JOURNAL OF CLINICAL INVESTIGATION (2010)
Molecular Architecture of the Human Sinus Node Insights Into the Function of the Cardiac Pacemaker
Natalie J. Chandler et al.
CIRCULATION (2009)
The RGK family of GTP-binding proteins: Regulators of voltage-dependent calcium channels and cytoskeleton remodeling
Robert N. Correll et al.
CELLULAR SIGNALLING (2008)
Calcium cycling protein density and functional importance to automaticity of isolated sinoatrial nodal cells are independent of cell size
Alexey E. Lyashkov et al.
CIRCULATION RESEARCH (2007)
Declining into failure - The age-dependent loss of the L-type calcium channel within the sinoatrial node
Sandra A. Jones et al.
CIRCULATION (2007)
The role of heart rate variability in prognosis for different modes of death in chronic heart failure
Gavin Richard H. Sandercock et al.
PACE-PACING AND CLINICAL ELECTROPHYSIOLOGY (2006)
Bradycardia and slowing of the atrioventricular conduction in mice lacking CaV3.1/α1G T-type calcium channels
ME Mangoni et al.
CIRCULATION RESEARCH (2006)
Regulation of voltage-gated calcium channel activity by the Rem and Rad GTPases
BS Finlin et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2003)
Functional role of L-type Cav13Ca2+ channels in cardiac pacemaker activity
ME Mangoni et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2003)
Sarcoplasmic reticulum Ca2+ release is not a dominating factor in sinoatrial node pacemaker activity
H Honjo et al.
CIRCULATION RESEARCH (2003)
Temporally regulated and tissue-specific gene manipulations in the adult and embryonic heart using a tamoxifen-inducible Cre protein
DS Sohal et al.
CIRCULATION RESEARCH (2001)
Expression of T- and L-type calcium channel mRNA in murine sinoatrial node
G Bohn et al.
FEBS LETTERS (2000)
The sinoatrial node, a heterogeneous pacemaker structure
MR Boyett et al.
CARDIOVASCULAR RESEARCH (2000)