Journal
INTERNAL MEDICINE
Volume 60, Issue 7, Pages 1019-1025Publisher
JAPAN SOC INTERNAL MEDICINE
DOI: 10.2169/internalmedicine.5837-20
Keywords
gastric polyp; hypergastrinemia; argon plasma coagulation; proton pump inhibitor; gastric antral vascular ectasia
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This case involves a patient with gastric antral vascular ectasia (GAVE) who developed gastric hyperplastic polyps after argon plasma coagulation (APC) therapy. The polyps and GAVE disappeared after discontinuing proton pump inhibitors (PPIs).
We herein report a case of gastric hyperplastic polyps after argon plasma coagulation (APC) for gastric antral vascular ectasia (GAVE) in the antrum of a 65-year-old man with liver cirrhosis and hypergastrinemia induced by long-term proton pump inhibitor (PPI) use. Two years after APC therapy, endoscopy demonstrated multiple gastric polyps in the antrum and angle. A gastric polyp biopsy indicated foveolar epithelium hyperplasia, which was diagnosed as gastric hyperplastic polyps. One year after switching to an H2 blocker antagonist, endoscopy revealed that the polyps and GAVE had disappeared, with normal gastrin levels suggesting that PPI-induced hypergastrinemia had caused gastric hyperplastic polyps after APC therapy, and the polyps had disappeared after discontinuing PPIs.
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