Related references
Note: Only part of the references are listed.Receptor for Advanced Glycation End Products (RAGE) Mediates Cognitive Impairment Triggered by Pneumococcal Meningitis
Vijayasree V. Giridharan et al.
NEUROTHERAPEUTICS (2021)
Androgens Increase Accumulation of Advanced Glycation End Products in Granulosa Cells by Activating ER Stress in PCOS
Jerilee M. K. Azhary et al.
ENDOCRINOLOGY (2020)
Diaphanous-1 affects the nanoscale clustering and lateral diffusion of receptor for advanced glycation endproducts (RAGE)
Qiaochu Zhu et al.
BIOCHIMICA ET BIOPHYSICA ACTA-BIOMEMBRANES (2019)
Insights into the effects of N-glycosylation on the characteristics of the VC1 domain of the human receptor for advanced glycation end products (RAGE) secreted by Pichia pastoris
Genny Degani et al.
GLYCOCONJUGATE JOURNAL (2019)
RAGE-NF-κB-PPARγ Signaling is Involved in AGEs-Induced Upregulation of Amyloid-β Influx Transport in an In Vitro BBB Model
Fang Chen et al.
NEUROTOXICITY RESEARCH (2018)
Advanced Glycation End Products Modulate Amyloidogenic APP Processing and Tau Phosphorylation: A Mechanistic Link between Glycation and the Development of Alzheimer's Disease
Kedar Batkulwar et al.
ACS CHEMICAL NEUROSCIENCE (2018)
Targeted inhibition of RAGE reduces amyloid-β influx across the blood-brain barrier and improves cognitive deficits in db/db mice
Hao Wang et al.
NEUROPHARMACOLOGY (2018)
Impaired oligodendrogenesis and myelination by elevated S100B levels during neurodevelopment
Gisela Santos et al.
NEUROPHARMACOLOGY (2018)
Free Radical Damage in Ischemia-Reperfusion Injury: An Obstacle in Acute Ischemic Stroke after Revascularization Therapy
Ming-Shuo Sun et al.
OXIDATIVE MEDICINE AND CELLULAR LONGEVITY (2018)
Neuroprotective effect of the group III mGlu receptor agonist ACPT-I after ischemic stroke in rats with essential hypertension
Helena Domin et al.
PROGRESS IN NEURO-PSYCHOPHARMACOLOGY & BIOLOGICAL PSYCHIATRY (2018)
AGEs, RAGEs and s-RAGE; friend or foe for cancer
Saheem Ahmad et al.
SEMINARS IN CANCER BIOLOGY (2018)
HMG-CoA Reductase Inhibitors Relieve Endoplasmic Reticulum Stress by Autophagy Inhibition in Rats With Permanent Brain Ischemia
Tao Zhang et al.
FRONTIERS IN NEUROSCIENCE (2018)
Liraglutide Ameliorates beta-Amyloid Deposits and Secondary Damage in the Ipsilateral Thalamus and Sensory Deficits After Focal Cerebral Infarction in Rats
Hui-Li Zhu et al.
FRONTIERS IN NEUROSCIENCE (2018)
Association of Long-Term Atorvastatin with Escalated Stroke-Induced Neuroinflammation in Rats
Leila Simani et al.
JOURNAL OF MOLECULAR NEUROSCIENCE (2017)
Modulation of cerebral RAGE expression following nitric oxide synthase inhibition in rats subjected to focal cerebral ischemia
Rosaria Greco et al.
EUROPEAN JOURNAL OF PHARMACOLOGY (2017)
Targeting the Receptor for Advanced Glycation Endproducts (RAGE): A Medicinal Chemistry Perspective
Salvatore Bongarzone et al.
JOURNAL OF MEDICINAL CHEMISTRY (2017)
Inhibition of AGEs/RAGE/Rho/ROCK.pathway suppresses non-specific neuroinflammation by regulating BV2 microglial M1/M2 polarization through the NF-κB pathway
Jingkao Chen et al.
JOURNAL OF NEUROIMMUNOLOGY (2017)
The Astrocytic S100B Protein with Its Receptor RAGE Is Aberrantly Expressed in SOD1G93A Models, and Its Inhibition Decreases the Expression of Proinflammatory Genes
Alessia Serrano et al.
MEDIATORS OF INFLAMMATION (2017)
RAGE-Specific Inhibitor FPS-ZM1 Attenuates AGEs-Induced Neuroinflammation and Oxidative Stress in Rat Primary Microglia
Chao Shen et al.
NEUROCHEMICAL RESEARCH (2017)
Homeostatic nuclear RAGE-ATM interaction is essential for efficient DNA repair
Varun Kumar et al.
NUCLEIC ACIDS RESEARCH (2017)
Advanced glycation end-products produced systemically and by macrophages: A common contributor to inflammation and degenerative diseases
Kyunghee Byun et al.
PHARMACOLOGY & THERAPEUTICS (2017)
The AGE-RAGE Axis: Implications for Age-Associated Arterial Diseases
Laura M. Senatus et al.
FRONTIERS IN GENETICS (2017)
Protective effect of mangiferin on myocardial ischemia-reperfusion injury in streptozotocin-induced diabetic rats: role of AGE-RAGE/MAPK pathways
Kapil Suchal et al.
SCIENTIFIC REPORTS (2017)
HMGB1 promotes neurovascular remodeling via Rage in the late phase of 0 subarachnoid hemorrhage
Xiaodi Tian et al.
BRAIN RESEARCH (2017)
Prevalence, Incidence, and Mortality of Stroke in China Results from a Nationwide Population-Based Survey of 480 687 Adults
Wenzhi Wang et al.
CIRCULATION (2017)
Neuroprotection in acute stroke: targeting excitotoxicity, oxidative and nitrosative stress, and infl ammation
Angel Chamorro et al.
LANCET NEUROLOGY (2016)
Effects of RAGE-Specific Inhibitor FPS-ZM1 on Amyloid-β Metabolism and AGEs-Induced Inflammation and Oxidative Stress in Rat Hippocampus
Yan Hong et al.
NEUROCHEMICAL RESEARCH (2016)
RAGE axis in neuroinflammation, neurodegeneration and its emerging role in the pathogenesis of amyotrophic lateral sclerosis
Rashmi Ray et al.
NEUROSCIENCE AND BIOBEHAVIORAL REVIEWS (2016)
Change in the Molecular Dimension of a RAGE-Ligand Complex Triggers RAGE Signaling
Jing Xue et al.
STRUCTURE (2016)
The Neuroprotection of Liraglutide Against Ischaemia-induced Apoptosis through the Activation of the PI3K/AKT and MAPK Pathways
Huili Zhu et al.
SCIENTIFIC REPORTS (2016)
Atorvastatin attenuates cognitive deficits through Akt1/caspase-3 signaling pathway in ischemic stroke
Jie Yang et al.
BRAIN RESEARCH (2015)
The RAGE receptor and its ligands are highly expressed in astrocytes in a grade-dependant manner in the striatum and subependymal layer in Huntington's disease
Joanne Kim et al.
JOURNAL OF NEUROCHEMISTRY (2015)
In Vivo Inhibition of miR-155 Promotes Recovery after Experimental Mouse Stroke
Ernesto Caballero-Garrido et al.
JOURNAL OF NEUROSCIENCE (2015)
Receptor for Advanced Glycation End-Product Antagonist Reduces Blood-Brain Barrier Damage After Intracerebral Hemorrhage
Fan Yang et al.
STROKE (2015)
Animal models of ischemic stroke and their application in clinical research
Felix Fluri et al.
DRUG DESIGN DEVELOPMENT AND THERAPY (2015)
Activation of the High-Mobility Group Box 1 Protein-Receptor for Advanced Glycation End-Products Signaling Pathway in Rats During Neurogenesis After Intracerebral Hemorrhage
Chunyan Lei et al.
STROKE (2015)
Neuroprotection by the PARP inhibitor PJ34 modulates cerebral and circulating RAGE levels in rats exposed to focal brain ischemia
Rosaria Greco et al.
EUROPEAN JOURNAL OF PHARMACOLOGY (2014)
S100B protein activates a RAGE-dependent autocrine loop in astrocytes: implications for its role in the propagation of reactive gliosis
Alejandro Villarreal et al.
JOURNAL OF NEUROCHEMISTRY (2014)
Optogenetic Mapping after Stroke Reveals Network-Wide Scaling of Functional Connections and Heterogeneous Recovery of the Peri-Infarct
Diana H. Lim et al.
JOURNAL OF NEUROSCIENCE (2014)
Effect of Transient Cerebral Ischemia on the Expression of Receptor for Advanced Glycation End Products (RAGE) in the Gerbil Hippocampus Proper
Jae-Chul Lee et al.
NEUROCHEMICAL RESEARCH (2014)
Aβ-AGE AGGRAVATES COGNITIVE DEFICIT IN RATS VIA RAGE PATHWAY
C. Chen et al.
NEUROSCIENCE (2014)
Oxidative Stress-Mediated Reperfusion Injury: Mechanism and Therapies
Zhengyuan Xia et al.
OXIDATIVE MEDICINE AND CELLULAR LONGEVITY (2014)
Guidelines for the Prevention of Stroke in Patients With Stroke and Transient Ischemic Attack A Guideline for Healthcare Professionals From the American Heart Association/American Stroke Association
Walter N. Kernan et al.
STROKE (2014)
Perspectives on RAGE Signaling and Its Role in Cardiovascular Disease
M. Michael Cohen
AMERICAN JOURNAL OF MEDICAL GENETICS PART A (2013)
Alternative Splicing of the RAGE Cytoplasmic Domain Regulates Cell Signaling and Function
Joel Jules et al.
PLOS ONE (2013)
Guidelines for the Early Management of Patients With Acute Ischemic Stroke A Guideline for Healthcare Professionals From the American Heart Association/American Stroke Association
Edward C. Jauch et al.
STROKE (2013)
Distal Occlusion of the Middle Cerebral Artery in Mice: Are We Ready to Assess Long-Term Functional Outcome?
Anna Rosell et al.
TRANSLATIONAL STROKE RESEARCH (2013)
Simvastatin attenuates axonal injury after experimental traumatic brain injury and promotes neurite outgrowth of primary cortical neurons
Hongtao Wu et al.
BRAIN RESEARCH (2012)
Formin mDia1 Mediates Vascular Remodeling via Integration of Oxidative and Signal Transduction Pathways
Fatouma Toure et al.
CIRCULATION RESEARCH (2012)
A multimodal RAGE-specific inhibitor reduces amyloid β-mediated brain disorder in a mouse model of Alzheimer disease
Rashid Deane et al.
JOURNAL OF CLINICAL INVESTIGATION (2012)
Modulation of RAGE Isoforms Expression in the Brain and Plasma of Rats Exposed to Transient Focal Cerebral Ischemia
Rosaria Greco et al.
NEUROCHEMICAL RESEARCH (2012)
RAGE mediates vascular injury and inflammation after global cerebral ischemia
Tomoya Kamide et al.
NEUROCHEMISTRY INTERNATIONAL (2012)
RAGE biology, atherosclerosis and diabetes
Drazenka Pongrac Barlovic et al.
CLINICAL SCIENCE (2011)
S100B Protein Stimulates Microglia Migration via RAGE-dependent Up-regulation of Chemokine Expression and Release
Roberta Bianchi et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
The G82S Polymorphism Promotes Glycosylation of the Receptor for Advanced Glycation End Products (RAGE) at Asparagine 81 COMPARISON OF WILD-TYPE RAGE WITH THE G82S POLYMORPHIC VARIANT
Sun Jin Park et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
S100B alters neuronal survival and dendrite extension via RAGE-mediated NF-κB signaling
Alejandro Villarreal et al.
JOURNAL OF NEUROCHEMISTRY (2011)
Tanshinone II A down-regulates HMGB1, RAGE, TLR4, NF-κB expression, ameliorates BBB permeability and endothelial cell function, and protects rat brains against focal ischemia
Lina Wang et al.
BRAIN RESEARCH (2010)
Inflammation in ischaemic brain injury: Current advances and future perspectives
Weiliang Xia et al.
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY (2010)
Homodimerization Is Essential for the Receptor for Advanced Glycation End Products (RAGE)-mediated Signal Transduction
Hongliang Zong et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2010)
S100B/RAGE-dependent activation of microglia via NF-κB and AP-1 Co-regulation of COX-2 expression by S100B, IL-1β and TNF-α
Roberta Bianchi et al.
NEUROBIOLOGY OF AGING (2010)
Soluble forms of RAGE in internal medicine
Natale Vazzana et al.
INTERNAL AND EMERGENCY MEDICINE (2009)
Levels of Soluble Receptor for Advanced Glycation End Products in Acute Ischemic Stroke without a Source of Cardioembolism
Hyun-Young Park et al.
JOURNAL OF CLINICAL NEUROLOGY (2009)
Neuronal RAGE expression modulates severity of injury following transient focal cerebral ischemia
Benjamin G. Hassid et al.
JOURNAL OF CLINICAL NEUROSCIENCE (2009)
Calcium-regulated intramembrane proteolysis of the RAGE receptor
Arnaud Galichet et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2008)
Oxygen deprivation triggers upregulation of early growth response-1 by the receptor for advanced glycation end products
Jong Sun Chang et al.
CIRCULATION RESEARCH (2008)
Interaction of the RAGE Cytoplasmic Domain with Diaphanous-1 Is Required for Ligand-stimulated Cellular Migration through Activation of Rac1 and Cdc42
Barry I. Hudson et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2008)
The HMGB1 Receptor RAGE Mediates Ischemic Brain Damage
Sajjad Muhammad et al.
JOURNAL OF NEUROSCIENCE (2008)
Nuclear factor-κB activation and postischemic inflammation are suppressed in CD36-null mice after middle cerebral artery occlusion
Alexander Kunz et al.
JOURNAL OF NEUROSCIENCE (2008)
RAGE expression is up-regulated in human cerebral ischemia and pMCAO rats
Dong-Xu Zhai et al.
NEUROSCIENCE LETTERS (2008)
Overexpression of human S100B exacerbates brain damage and periinfarct gliosis after permanent focal ischemia
Takashi Mori et al.
STROKE (2008)
Early release of HMGB-1 from neurons after the onset of brain ischemia
Jianhua Qiu et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2008)
Hypoxia-inducible factor-1 mediates neuronal expression of the receptor for advanced glycation end products following hypoxia/ischemia
Paola Pichiule et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2007)
De-N-glycosylation or G82S mutation of RAGE sensitizes its interaction with advanced glycation endproducts
Marl Osawa et al.
BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS (2007)
Evaluation of rage isoforms, ligands, and signaling in the brain
QX Ding et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR CELL RESEARCH (2005)
Splice variants of the receptor for advanced glycosylation end products (RAGE) in human brain
QX Ding et al.
NEUROSCIENCE LETTERS (2005)
Neuronal activation of NF-kappa B contributes to cell death in cerebral ischemia
W Zhang et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2005)
Spermine reduces infarction and neurological deficit following a rat model of middle cerebral artery occlusion: A magnetic resonance imaging study
M Shirhan et al.
NEUROSCIENCE (2004)
In Situ Detection of Neuronal DNA Strand Breaks Using the Klenow Fragment of DNA Polymerase I Reveals Different Mechanisims of Neuron Death After Global Cerebral Ischemia
Kunlin Jin et al.
JOURNAL OF NEUROCHEMISTRY (2003)
RAGE is expressed in pyramidal cells of the hippocampus following moderate hypoxic-ischemic brain injury in rats
L Ma et al.
BRAIN RESEARCH (2003)
Novel splice variants of the receptor for advanced glycation end-products expressed in human vascular endothelial cells and pericytes, and their putative roles in diabetes-induced vascular injury
H Yonekura et al.
BIOCHEMICAL JOURNAL (2003)
Triflusal posttreatment inhibits glial nuclear factor-κB, downregulates the glial response, and is neuroprotective in an excitotoxic injury model in postnatal brain
L Acarin et al.
STROKE (2001)
The receptor for advanced glycation end products is induced by the glycation products themselves and tumor necrosis factor-α through nuclear Factor-κB, and by 17β-estradiol through Sp-1 in human vascular endothelial cells
N Tanaka et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)
Transcription factor nuclear factor-kappa B is activated in neurons after focal cerebral ischemia
D Stephenson et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2000)
Share Paper
