Journal
CURRENT OPINION IN PHARMACOLOGY
Volume 29, Issue -, Pages 1-6Publisher
ELSEVIER SCI LTD
DOI: 10.1016/j.coph.2016.03.002
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Funding
- Italian Association for Cancer Research [AIRC: IG-14442]
- University of Ferrara, Telethon [GGP15219/B]
- Italian Cystic Fibrosis Foundation (FFC) [19/2014]
- Italian Ministry of Health
- Italian Ministry of Education, University and Research [20129JLHSY_002, RBAP11FXBC_002, RBFR10EGVP_001]
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Typical hallmarks of cancer include programmed cell death evasion, uncontrolled cell growth, invasion, and metastasis. Changes in intracellular Ca2+ levels can modulate signaling pathways that control a broad range of cellular events, including those important to tumorigenesis and cancer progression. Here we discuss how known molecular mediators of cellular Ca2+ homeostasis impact tumor dynamics and how deregulation of major oncogenes and tumor suppressors is tightly associated with Ca2+ signaling.
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