Related references
Note: Only part of the references are listed.The deubiquitinase USP7 stabilizes Maf proteins to promote myeloma cell survival
Yuanming He et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2020)
Lanatoside C inhibits cell proliferation and induces apoptosis through attenuating Wnt/β-catenin/c-Myc signaling pathway in human gastric cancer cell
Yudong Hu et al.
BIOCHEMICAL PHARMACOLOGY (2018)
The transmembrane protein TMEPAI induces myeloma cell apoptosis by promoting degradation of the c-Maf transcription factor
Yanyun Du et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2018)
OTUB1 protein suppresses mTOR complex 1 (mTORC1) activity by deubiquitinating the mTORC1 inhibitor DEPTOR
Linlin Zhao et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2018)
OTUB1 promotes esophageal squamous cell carcinoma metastasis through modulating Snail stability
Honghong Zhou et al.
ONCOGENE (2018)
The ubiquitin-conjugating enzyme UBE2O modulates c-Maf stability and induces myeloma cell apoptosis
Yujia Xu et al.
JOURNAL OF HEMATOLOGY & ONCOLOGY (2017)
Inhibition of the deubiquitinase USP5 leads to c-Maf protein degradation and myeloma cell apoptosis
Siyu Wang et al.
CELL DEATH & DISEASE (2017)
Ring finger protein 6 promotes breast cancer cell proliferation by stabilizing estrogen receptor alpha
Yuanying Zeng et al.
ONCOTARGET (2017)
OTUB1 inhibits the ubiquitination and degradation of FOXM1 in breast cancer and epirubicin resistance
U. Karunarathna et al.
ONCOGENE (2016)
The ubiquitin ligase HERC4 mediates c-Maf ubiquitination and delays the growth of multiple myeloma xenografts in nude mice
Zubin Zhang et al.
BLOOD (2016)
MAF protein mediates innate resistance to proteasome inhibition therapy in multiple myeloma
Ya-Wei Qiang et al.
BLOOD (2016)
OTUB1 triggers lung cancer development by inhibiting RAS monoubiquitination
Maria Francesca Baietti et al.
EMBO MOLECULAR MEDICINE (2016)
Liver cancer cells are sensitive to Lanatoside C induced cell death independent of their PTEN status
Irem Durmaz et al.
PHYTOMEDICINE (2016)
Lanatoside C suppressed colorectal cancer cell growth by inducing mitochondrial dysfunction and increased radiation sensitivity by impairing DNA damage repair
Mi Ae Kang et al.
ONCOTARGET (2016)
PROGgeneV2: enhancements on the existing database
Chirayu Pankaj Goswami et al.
BMC CANCER (2014)
Widespread Genetic Heterogeneity in Multiple Myeloma: Implications for Targeted Therapy
Jens G. Lohr et al.
CANCER CELL (2014)
Ubiquitination of the transcription factor c-MAF is mediated by multiple lysine residues
Guodong Chen et al.
INTERNATIONAL JOURNAL OF BIOCHEMISTRY & CELL BIOLOGY (2014)
International Myeloma Working Group updated criteria for the diagnosis of multiple myeloma
S. Vincent Rajkumar et al.
LANCET ONCOLOGY (2014)
OTUB1 modulates c-IAP1 stability to regulate signalling pathways
Tatiana Goncharov et al.
EMBO JOURNAL (2013)
OTUB1 enhances TGFβ signalling by inhibiting the ubiquitylation and degradation of active SMAD2/3
Lina Herhaus et al.
NATURE COMMUNICATIONS (2013)
The mechanism of OTUB1-mediated inhibition of ubiquitination
Reuven Wiener et al.
NATURE (2012)
Lanatoside C sensitizes glioblastoma cells to tumor necrosis factor-related apoptosis-inducing ligand and induces an alternative cell death pathway
Christian E. Badr et al.
NEURO-ONCOLOGY (2011)
A SNP Microarray and FISH-Based Procedure to Detect Allelic Imbalances in Multiple Myeloma: An Integrated Genomics Approach Reveals a Wide Gene Dosage Effect
Luca Agnelli et al.
GENES CHROMOSOMES & CANCER (2009)
A chemical biology screen identifies glucocorticoids that regulate c-maf expression by increasing its proteasomal. degradation through up-regulation of ubiquitin
Xinliang Mao et al.
BLOOD (2007)
Gene-expression signature of benign monoclonal gammopathy evident in multiple myeloma is linked to good prognosis
Fenghuang Zhan et al.
BLOOD (2007)
The molecular classification of multiple myeloma
Fenghuang Zhan et al.
BLOOD (2006)
ARK5 is transcriptionally regulated by the Large-MAF family and mediates IGF-1-induced cell invasion in multiple myeloma: ARK5 as a new molecular determinant of malignant multiple myeloma
A Suzuki et al.
ONCOGENE (2005)
Overexpression of c-maf is a frequent oncogenic event in multiple myeloma that promotes proliferation and pathological interactions with bone marrow stroma
EM Hurt et al.
CANCER CELL (2004)
Share Paper
