Journal
CELL METABOLISM
Volume 31, Issue 6, Pages 1047-1049Publisher
CELL PRESS
DOI: 10.1016/j.cmet.2020.05.010
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Funding
- American Heart Association [19PRE34380515]
- National Institutes of Health (NIH)/National Institute of General Medical Sciences (NIGMS) Cellular, Biochemical, and Molecular Sciences (CBMS) T32
- NIH/NIGMS [1R35GM128915-01]
- NIH/National Cancer Institute [1R21 CA227483-01A1]
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Mitochondrial fission is sustained through contact with several organelles, including the endoplasmic reticulum, lysosomes, and the actin cytoskeleton. Nagashima et al. (2020) now demonstrate that PI(4)P-containing Golgi-derived vesicles also modulate mitochondrial fission, driven by Arf1 and PI(4)KIII beta activity, identifying a new organelle contact involved in maintaining mitochondrial homeostasis.
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