Journal
ACS CHEMICAL NEUROSCIENCE
Volume 11, Issue 15, Pages 2156-2158Publisher
AMER CHEMICAL SOC
DOI: 10.1021/acschemneuro.0c00422
Keywords
COVID-19; Cytokine release syndrome; Interstitial pulmonary edema; Acute disseminated encephalomyelitis; Thromboembolic disease
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Lung injury with COVID-19 may be due to a complex underlying pathophysiology. Cytokine release syndrome appears to be a catalyst of different inflammatory pathways promoting lung parenchymal injury and thromboembolic phenomena (dual hit injury). Recently, severe neurological manifestations such as acute disseminated encephalomyelitis, which may be not linked to lung pathology, have been identified in COVID-19, contributing thus further to the versatility of its clinical features.
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