Journal
NATURE NEUROSCIENCE
Volume 23, Issue 5, Pages 596-597Publisher
NATURE PORTFOLIO
DOI: 10.1038/s41593-020-0622-x
Keywords
-
Categories
Ask authors/readers for more resources
At the heart of C9ORF72-related amyotrophic lateral sclerosis and frontotemporal dementia (ALS /FTD) research lies the mechanistic question of whether disease is caused by toxic gain of function related to the repeat expansion, loss of endogenous C9ORF72 expression, or both. New findings provide insights to this question.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available