4.7 Editorial Material

Mitochondria Matter: A Critical Role of ADCK4 in Stabilizing the CoQ Complex in Podocytes in Steroid-Resistant Nephrotic Syndrome

Related references

Note: Only part of the references are listed.
Article Urology & Nephrology

ADCK4 Deficiency Destabilizes the Coenzyme Q Complex, Which Is Rescued by 2,4-Dihydroxybenzoic Acid Treatment

Eugen Widmeier et al.

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2020)

Article Medicine, Research & Experimental

ATP-binding cassette A1 deficiency causes cardiolipin-driven mitochondrial dysfunction in podocytes

G. Michelle Ducasa et al.

JOURNAL OF CLINICAL INVESTIGATION (2019)

Article Urology & Nephrology

Deficient Autophagy Results in Mitochondrial Dysfunction and FSGS

Takahisa Kawakami et al.

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2015)

Article Physiology

Mpv17 in mitochondria protects podocytes against mitochondrial dysfunction and apoptosis in vivo and in vitro

Gabriela Casalena et al.

AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2014)

Article Medicine, Research & Experimental

ADCK4 mutations promote steroid-resistant nephrotic syndrome through CoQ10 biosynthesis disruption

Shazia Ashraf et al.

JOURNAL OF CLINICAL INVESTIGATION (2013)

Article Urology & Nephrology

A High-Powered View of the Filtration Barrier

Janos Peti-Peterdi et al.

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2010)

Article Multidisciplinary Sciences

Restoration of electron transport without proton pumping in mammalian mitochondria

Ester Perales-Clemente et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)

Review Urology & Nephrology

The genetic basis of FSGS and steroid-resistant nephrosis

MR Pollak

SEMINARS IN NEPHROLOGY (2003)