Journal
IMMUNITY
Volume 52, Issue 2, Pages 207-209Publisher
CELL PRESS
DOI: 10.1016/j.immuni.2020.01.011
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Funding
- Crohn's and Colitis Foundation fellowship [608975]
- National Institutes of Health (NIH) [R01AI143842, R01AI123368, R01AI145989, R21CA249274, U01AI095608]
- NIAID Mucosal Immunology Studies Team (MIST)
- Searle Scholars Program
- American Asthma Foundation Scholar Award
- Burroughs Wellcome Fund
- Wade F.B. Thompson/Cancer Research Institute (CRI) CLIP Investigator grant
- Meyer Cancer Center Collaborative Research Initiative
- Roberts Institute for Research in IBD
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Innate lymphoid cells (ILCs) are tissue-resident lymphocytes that promote immunity to pathogens at mucosal barriers, but the mechanisms regulating their development within tissues remain poorly understood. In this issue of Immunity, Oherle et al. identify a niche in the neonatal airway where stromal cell-derived insulin-like growth factor 1 (IGF1) supports the proliferation of ILC precursors and protects from infection.
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