Related references
Note: Only part of the references are listed.MiR-451a suppressing BAP31 can inhibit proliferation and increase apoptosis through inducing ER stress in colorectal cancer
Ke Xu et al.
CELL DEATH & DISEASE (2019)
STX17 dynamically regulated by Fis1 induces mitophagy via hierarchical macroautophagic mechanism
Hongxu Xian et al.
NATURE COMMUNICATIONS (2019)
BAP31 regulates mitochondrial function via interaction with Tom40 within ER-mitochondria contact sites
Takushi Namba
SCIENCE ADVANCES (2019)
Inhibition of BAP31 expression inhibits cervical cancer progression by suppressing metastasis and inducing intrinsic and extrinsic apoptosis
An Wang et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2019)
BAP31, a newly defined cancer/testis antigen, regulates proliferation, migration, and invasion to promote cervical cancer progression
Erle Dang et al.
CELL DEATH & DISEASE (2018)
Microenvironmental autophagy promotes tumour growth
Nadja S. Katheder et al.
NATURE (2017)
Legionella effector Lpg1137 shuts down ER-mitochondria communication through cleavage of syntaxin 17
Kohei Arasaki et al.
NATURE COMMUNICATIONS (2017)
ER stress: Autophagy induction, inhibition and selection
Harun-Or Rashid et al.
AUTOPHAGY (2015)
ATG14 promotes membrane tethering and fusion of autophagosomes to endolysosomes
Jiajie Diao et al.
NATURE (2015)
Zidovudine, an anti-viral drug, resensitizes gemcitabine-resistant pancreatic cancer cells to gemcitabine by inhibition of the Akt-GSK3β-Snail pathway
T. Namba et al.
CELL DEATH & DISEASE (2015)
Endoplasmic Reticulum Stress in Malignancy
Hanna J. Clarke et al.
CANCER CELL (2014)
ER-associated degradation: Protein quality control and beyond
Annamaria Ruggiano et al.
JOURNAL OF CELL BIOLOGY (2014)
Metabolic control of cell death
Douglas R. Green et al.
SCIENCE (2014)
Autophagosomes form at ER-mitochondria contact sites
Maho Hamasaki et al.
NATURE (2013)
The autophagosome: origins unknown, biogenesis complex
Christopher A. Lamb et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2013)
CDIP1-BAP31 Complex Transduces Apoptotic Signals from Endoplasmic Reticulum to Mitochondria under Endoplasmic Reticulum Stress
Takushi Namba et al.
CELL REPORTS (2013)
Fis1 and Bap31 bridge the mitochondria-ER interface to establish a platform for apoptosis induction
Ryota Iwasawa et al.
EMBO JOURNAL (2011)
BAP31 and BiP are essential for dislocation of SV40 from the endoplasmic reticulum to the cytosol
Roger Geiger et al.
NATURE CELL BIOLOGY (2011)
Novel targets for endoplasmic reticulum stress-induced apoptosis in B-CLL
Emanuela Rosati et al.
BLOOD (2010)
BAP31 interacts with Sec61 translocons and promotes retrotranslocation of CFTRΔF508 via the Derlin-1 complex
Bing Wang et al.
CELL (2008)
Autophagy in the pathogenesis of disease
Beth Levine et al.
CELL (2008)
Bap31 Is a Novel Target of the Human Papillomavirus E5 Protein
Jennifer A. Regan et al.
JOURNAL OF VIROLOGY (2008)
The M2 splice isoform of pyruvate kinase is important for cancer metabolism and tumour growth
Heather R. Christofk et al.
NATURE (2008)
Endoplasmic reticulum stress-induced apoptosis - Multiple pathways and activation of p53-up-regulated modulator of apoptosis (PUMA) and NOXA by p53
JZ Li et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Caspase cleavage product of BAP31 induces mitochondrial fission through endoplasmic reticulum calcium signals, enhancing cytochrome c release to the cytosol
DG Breckenridge et al.
JOURNAL OF CELL BIOLOGY (2003)
The procaspase-8 isoform, procaspase-8L, recruited to the BAP31 complex at the endoplasmic reticulum
DG Breckenridge et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)