4.6 Article

Loss of adenomatous polyposis coli function renders intestinal epithelial cells resistant to the cytokine IL-22

Related references

Note: Only part of the references are listed.
Article Cell Biology

Chir99021 and Valproic acid reduce the proliferative advantage of Apc mutant cells

Alistair J. Langlands et al.

CELL DEATH & DISEASE (2018)

Review Biochemistry & Molecular Biology

Crosstalk between DNA Damage and Inflammation in the Multiple Steps of Carcinogenesis

Shosuke Kawanishi et al.

INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES (2017)

Article Multidisciplinary Sciences

Quantitative proteomics identifies STEAP4 as a critical regulator of mitochondrial dysfunction linking inflammation and colon cancer

Xiang Xue et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2017)

Article Oncology

LRG1 modulates epithelial-mesenchymal transition and angiogenesis in colorectal cancer via HIF-1α activation

Jingjing Zhang et al.

JOURNAL OF EXPERIMENTAL & CLINICAL CANCER RESEARCH (2016)

Review Immunology

Interleukin-22: Immunobiology and Pathology

Jarrod A. Dudakov et al.

ANNUAL REVIEW OF IMMUNOLOGY VOL 33 (2015)

Article Gastroenterology & Hepatology

Th22 cell accumulation is associated with colorectal cancer development

Yong-Hong Huang et al.

WORLD JOURNAL OF GASTROENTEROLOGY (2015)

Article Biochemistry & Molecular Biology

Glycogen Synthase Kinase-3β Stabilizes the Interleukin (IL)-22 Receptor from Proteasomal Degradation in Murine Lung Epithelia

Nathaniel M. Weathington et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2014)

Article Immunology

Innate lymphoid cells sustain colon cancer through production of interleukin-22 in a mouse model

Stefanie Kirchberger et al.

JOURNAL OF EXPERIMENTAL MEDICINE (2013)

Article Cell Biology

The beta-Catenin Destruction Complex

Jennifer L. Stamos et al.

COLD SPRING HARBOR PERSPECTIVES IN BIOLOGY (2013)

Article Multidisciplinary Sciences

IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine

Samuel Huber et al.

NATURE (2012)

Article Multidisciplinary Sciences

Adenoma-linked barrier defects and microbial products drive IL-23/IL-17-mediated tumour growth

Sergei I. Grivennikov et al.

NATURE (2012)

Article Biochemical Research Methods

Fiji: an open-source platform for biological-image analysis

Johannes Schindelin et al.

NATURE METHODS (2012)

Article Immunology

STAT3 links IL-22 signaling in intestinal epithelial cells to mucosal wound healing

Geethanjali Pickert et al.

JOURNAL OF EXPERIMENTAL MEDICINE (2009)

Article Biochemistry & Molecular Biology

Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

Yan Zheng et al.

NATURE MEDICINE (2008)

Article Cell Biology

Loss of APC induces polyploidy as a result of a combination of defects in mitosis and apoptosis

Dina Dikovskaya et al.

JOURNAL OF CELL BIOLOGY (2007)

Article Gastroenterology & Hepatology

IL-22 is increased in active Crohn's disease and promotes proinflammatory gene expression and intestinal epithelial cell migration

S Brand et al.

AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY (2006)

Review Immunology

Interleukin-22 activates STAT3 and induces IL-10 by colon epithelial cells

ML Nagalakshmi et al.

INTERNATIONAL IMMUNOPHARMACOLOGY (2004)

Article Biochemistry & Molecular Biology

Functional interaction of STAT3 transcription factor with the coactivator NcoA/SRC1a

S Giraud et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2002)

Article Immunology

Bacterial infection promotes colon tumorigenesis in ApcMin/+ mice

JV Newman et al.

JOURNAL OF INFECTIOUS DISEASES (2001)

Article Biochemistry & Molecular Biology

Interleukin (IL)-22, a novel human cytokine that signals through the interferon receptor-related proteins CRF2-4 and IL-22R

MH Xie et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2000)

Article Multidisciplinary Sciences

APC mutations in sporadic colorectal tumors:: A mutational hotspot and interdependence of the two hits

AJ Rowan et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)