Related references
Note: Only part of the references are listed.Carabin Protects Against Cardiac Hypertrophy by Blocking Calcineurin, Ras, and Ca2+/Calmodulin-Dependent Protein Kinase II Signaling
Malik Bisserier et al.
CIRCULATION (2015)
Dual-Specificity Phosphatase 14 (DUSP14/MKP6) Negatively Regulates TCR Signaling by Inhibiting TAB1 Activation
Chia-Yu Yang et al.
JOURNAL OF IMMUNOLOGY (2014)
BAMBI (BMP and activin membrane-bound inhibitor) protects the murine heart from pressure-overload biomechanical stress by restraining TGF-β signaling
Ana V. Villar et al.
BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2013)
The Dual-specificity Phosphatase DUSP14 Negatively Regulates Tumor Necrosis Factor- and Interleukin-1-induced Nuclear Factor-κB Activation by Dephosphorylating the Protein Kinase TAK1
Hao Zheng et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2013)
Potential of gene therapy as a treatment for heart failure
Roger J. Hajjar
JOURNAL OF CLINICAL INVESTIGATION (2013)
TAK1 Inhibition Promotes Apoptosis in KRAS-Dependent Colon Cancers
Anurag Singh et al.
CELL (2012)
The Progression of Hypertensive Heart Disease
Mark H. Drazner
CIRCULATION (2011)
Pivotal role of cardiomyocyte TGF-β signaling in the murine pathological response to sustained pressure overload
Norimichi Koitabashi et al.
JOURNAL OF CLINICAL INVESTIGATION (2011)
Epidemiology and risk profile of heart failure
Anh L. Bui et al.
NATURE REVIEWS CARDIOLOGY (2011)
Mitogen-Activated Protein Kinase Signaling in the Heart: Angels Versus Demons in a Heart-Breaking Tale
Beth A. Rose et al.
PHYSIOLOGICAL REVIEWS (2010)
Regulator of G protein signaling 5 protects against cardiac hypertrophy and fibrosis during biomechanical stress of pressure overload
Hongliang Li et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2010)
Dual-specificity phosphatases: critical regulators with diverse cellular targets
Kate I. Patterson et al.
BIOCHEMICAL JOURNAL (2009)
Characteristics of dual specificity phosphatases mRNA regulation by 3,4-methylenedioxymethamphetamine acute treatment in mice striatum
Cynthia Marie-Claire et al.
BRAIN RESEARCH (2008)
Left ventricular mass predicts heart failure not related to previous myocardial infarction: the Cardiovascular Health Study
Giovanni de Simone et al.
EUROPEAN HEART JOURNAL (2008)
Mechanisms of disease: Cardiac plasticity
Joseph A. Hill et al.
NEW ENGLAND JOURNAL OF MEDICINE (2008)
Increasing GLP-1-induced β-cell proliferation by silencing the negative regulators of signaling cAMP response element modulator-α and DUSP14
Sonia Klinger et al.
DIABETES (2008)
Inhibition of apoptosis-regulated signaling kinase-1 and prevention of congestive heart failure by estrogen
Minoru Satoh et al.
CIRCULATION (2007)
Targeting dual-specificity phosphatases: manipulating MAP kinase signalling and immune responses
Kate L. Jeffrey et al.
NATURE REVIEWS DRUG DISCOVERY (2007)
Regulation of cardiac hypertrophy by intracellular signalling pathways
Joerg Heineke et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2006)
Activation of TGF-β1-TAK1-p38 MAPK pathway in spared cardiomyocytes is involved in left ventricular remodeling after myocardial infarction in rats
M Matsumoto-Ida et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2006)
TAK1, but not TAB1 or TAB2, plays an essential role in multiple signaling pathways in vivo
JH Shim et al.
GENES & DEVELOPMENT (2005)
Thyroid hormone induces cardiac myocyte hypertrophy in a thyroid hormone receptor α1-specific manner that requires TAK1 and p38 mitogen-activated protein kinase
K Kinugawa et al.
MOLECULAR ENDOCRINOLOGY (2005)
Increased left ventricular mass is a risk factor for the development of a depressed left ventricular ejection fraction within five years
MH Drazner et al.
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY (2004)
c-Jun N-terminal kinases (JNK) antagonize cardiac growth through cross-talk with calcineurin-NFAT signaling
QR Liang et al.
EMBO JOURNAL (2003)
Targeted inhibition of p38 MAPK promotes hypertrophic cardiomyopathy through upregulation of calcineurin-NEAT signaling
JC Braz et al.
JOURNAL OF CLINICAL INVESTIGATION (2003)
MEKK1 is essential for cardiac hypertrophy and dysfunction induced by Gq
T Minamino et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
Activated glycogen synthase-3β suppresses cardiac hypertrophy in vivo
CL Antos et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2002)
Redox regulation of MAPK pathways and cardiac hypertrophy in adult rat cardiac myocyte
K Tanaki et al.
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY (2001)
Negative-feedback regulation of CD28 costimulation by a novel mitogen-activated protein kinase phosphatase, MKP6
F Marti et al.
JOURNAL OF IMMUNOLOGY (2001)
TAK1 is activated in the myocardium after pressure overload and is sufficient to provoke heart failure in transgenic mice
D Zhang et al.
NATURE MEDICINE (2000)
Share Paper
