4.7 Article

Core pluripotency factors promote glycolysis of human embryonic stem cells by activating GLUT1 enhancer

Journal

PROTEIN & CELL
Volume 10, Issue 9, Pages 668-680

Publisher

SPRINGEROPEN
DOI: 10.1007/s13238-019-0637-9

Keywords

human embryonic stem cell; pluripotency factors; metabolism; Glut1; enhancer; promoter; epigenetics; chromosome interaction

Categories

Funding

  1. National Natural Science Foundation of China [81430032, U1601222]
  2. Guangdong Province Program [00201516]
  3. Key Research and Development Program of Guangdong Province [2019B020235003]
  4. Major basic research developmental project of the Natural Science Foundation of Guangdong Province [2014A030308018]
  5. Science and Technology Innovation Committee of Shenzhen Municipality [JCYJ20180504170301309]
  6. Shenzhen Sanming Project of Medicine [SZSM201602102]
  7. Guangdong Provincial Key Laboratory of Cancer Immunotherapy

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Human embryonic stem cells (hESCs) depend on glycolysis for energy and substrates for biosynthesis. To understand the mechanisms governing the metabolism of hESCs, we investigated the transcriptional regulation of glucose transporter 1 (GLUT1, SLC2A1), a key glycolytic gene to maintain pluripotency. By combining the genome-wide data of binding sites of the core pluripotency factors (SOX2, OCT4, NANOG, denoted SON), chromosomal interaction and histone modification in hESCs, we identified a potential enhancer of the GLUT1 gene in hESCs, denoted GLUT1 enhancer (GE) element. GE interacts with the promoter of GLUT1, and the deletion of GE significantly reduces the expression of GLUT1, glucose uptake and glycolysis of hESCs, confirming that GE is an enhancer of GLUT1 in hESCs. In addition, the mutation of SON binding motifs within GE reduced the expression of GLUT1 as well as the interaction between GE and GLUT1 promoter, indicating that the binding of SON to GE is important for its activity. Therefore, SON promotes glucose uptake and glycolysis in hESCs by inducing GLUT1 expression through directly activating the enhancer of GLUT1.

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