Related references
Note: Only part of the references are listed.Forced exercise-induced osteoarthritis is attenuated in mice lacking the small leucine-rich proteoglycan decorin
Tobias Gronau et al.
ANNALS OF THE RHEUMATIC DISEASES (2017)
Stimulation of Superficial Zone Protein/Lubricin/PRG4 by Transforming Growth Factor-β in Superficial Zone Articular Chondrocytes and Modulation by Glycosaminoglycans
Araceli Cuellar et al.
TISSUE ENGINEERING PART A (2015)
Deletion of the Transforming Growth Factor β Receptor Type II Gene in Articular Chondrocytes Leads to a Progressive Osteoarthritis-like Phenotype in Mice
Jie Shen et al.
ARTHRITIS AND RHEUMATISM (2013)
Inhibition of TGF-β signaling in mesenchymal stem cells of subchondral bone attenuates osteoarthritis
Gehua Zhen et al.
NATURE MEDICINE (2013)
Osteoarthritis: A disease of the joint as an organ
Richard F. Loeser et al.
ARTHRITIS AND RHEUMATISM (2012)
Chondrocyte-intrinsic Smad3 represses runx2-inducible matrix metalloproteinase 13 expression to maintain articular cartilage and prevent osteoarthritis
Carol G. Chen et al.
ARTHRITIS AND RHEUMATISM (2012)
Phenotypic spectrum of the SMAD3-related aneurysms-osteoarthritis syndrome
Ingrid M. B. H. van de Laar et al.
JOURNAL OF MEDICAL GENETICS (2012)
Mutations in SMAD3 cause a syndromic form of aortic aneurysms and dissections with early-onset osteoarthritis
Ingrid M. B. H. van de laar et al.
NATURE GENETICS (2011)
Genetic Variation in the SMAD3 Gene Is Associated With Hip and Knee Osteoarthritis
Ana M. Valdes et al.
ARTHRITIS AND RHEUMATISM (2010)
TGF-β/Smad3 signals repress chondrocyte hypertrophic differentiation and are required for maintaining articular cartilage
X Yang et al.
JOURNAL OF CELL BIOLOGY (2001)
Share Paper
