Journal
NEURON
Volume 101, Issue 6, Pages 991-993Publisher
CELL PRESS
DOI: 10.1016/j.neuron.2019.03.007
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Funding
- NINDS NIH HHS [R01 NS104386] Funding Source: Medline
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Exactly how cerebrovascular alterations contribute to Alzheimer's disease (AD) is still unknown. Merlini et al. (2019) show that blood-derived fibrinogen leads to dendritic spine elimination and cognitive deficit via microglial CD11b/CD18. Fibrinogen may be a significant contributor to AD pathogenesis.
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