Related references
Note: Only part of the references are listed.A novel nonsense mutation in the DMP1 gene in a Japanese family with autosomal recessive hypophosphatemic rickets
Ryusuke Koshida et al.
JOURNAL OF BONE AND MINERAL METABOLISM (2010)
Long-Term Clinical Outcome and Carrier Phenotype in Autosomal Recessive Hypophosphatemia Caused by a Novel DMP1 Mutation
Outi Makitie et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2010)
Fibroblast growth factor 23 production in bone is directly regulated by 1α,25-dihydroxyvitamin D, but not PTH
Fumie Saji et al.
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2010)
PTH increases FGF23 gene expression and mediates the high-FGF23 levels of experimental kidney failure: a bone parathyroid feedback loop
Vardit Lavi-Moshayoff et al.
AMERICAN JOURNAL OF PHYSIOLOGY-RENAL PHYSIOLOGY (2010)
Molecular analysis of DMP1 mutants causing autosomal recessive hypophosphatemic rickets
Emily G. Farrow et al.
BONE (2009)
Therapeutic Effects of Anti-FGF23 Antibodies in Hypophosphatemic Rickets/Osteomalacia
Yukiko Aono et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2009)
Regulation of Fibroblast Growth Factor 23 Production in Bone in Uremic Rats
Fumie Saji et al.
NEPHRON PHYSIOLOGY (2009)
Overexpression of fibroblast growth factor 23 suppresses osteoblast differentiation and matrix mineralization in vitro
Hua Wang et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2008)
Genetic Evidence of Serum Phosphate-Independent Functions of FGF-23 on Bone
Despina Sitara et al.
PLOS GENETICS (2008)
Regulation of osteoclast differentiation and function by phosphate: Potential role of osteoclasts in the skeletal abnormalities in hypophosphaternic conditions
Tetsuyuki Hayashibara et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2007)
Role of hyperphosphatemia and 1,25-dihydroxyvitamin D in vascular calcification and mortality in fibroblastic growth factor 23 null mice
Jason R. Stubbs et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2007)
Ablation of vitamin D signaling rescues bone, mineral, and glucose homeostasis in Fgf-23 deficient mice
Martina Hesse et al.
MATRIX BIOLOGY (2007)
Genetic ablation of vitamin D activation pathway reverses biochemical and skeletal anomalies in Fgf-23-null animals
Despina Sitara et al.
AMERICAN JOURNAL OF PATHOLOGY (2006)
DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis
Bettina Lorenz-Depiereux et al.
NATURE GENETICS (2006)
Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism
Jian Q. Feng et al.
NATURE GENETICS (2006)
Regulation of C-terminal and intact FGF-23 by dietary phosphate in men and women
Sherri-Ann M. Burnett et al.
JOURNAL OF BONE AND MINERAL RESEARCH (2006)
Pathogenic role of Fgf23 in Hyp mice
Shiguang Liu et al.
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2006)
Fibroblast growth factor 23 is a counter-regulatory phosphaturic hormone for vitamin D
Shiguang Liu et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2006)
Regulation of fibroblast growth factor-23 signaling by Klotho
H Kurosu et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Dietary and serum phosphorus regulate fibroblast growth factor 23 expression and 1,25-dihydroxyvitamin D metabolism in mice
F Perwad et al.
ENDOCRINOLOGY (2005)
Genetic dissection of phosphate- and vitamin D-mediated regulation of circulating Fgf23 concentrations
XJ Yu et al.
BONE (2005)
Vitamin D and phosphate regulate fibroblast growth factor-23 in K-562 cells
M Ito et al.
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2005)
Unique coexpression in osteoblasts of broadly expressed genes accounts for the spatial restriction of ECM mineralization to bone
M Murshed et al.
GENES & DEVELOPMENT (2005)
Circulating FGF-23 is regulated by 1α,25-dihydroxyvitamin D3 and phosphorus in vivo
H Saito et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
FGF-23 transgenic mice demonstrate hypophosphatemic rickets with reduced expression of sodium phosphate cotransporter type IIa
T Shimada et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2004)
Targeted ablation of Fgf23 demonstrates an essential physiological role of FGF23 in phosphate and vitamin D metabolism
T Shimada et al.
JOURNAL OF CLINICAL INVESTIGATION (2004)
FGF23 is processed by proprotein convertases but not by PHEX
A Beret-Pagès et al.
BONE (2004)
Transgenic mice expressing fibroblast growth factor 23 under the control of the α1(I) collagen promoter exhibit growth retardation, osteomalacia, and disturbed phosphate homeostasis
T Larsson et al.
ENDOCRINOLOGY (2004)
Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice
D Sitara et al.
MATRIX BIOLOGY (2004)
Transgenic mice overexpressing human fibroblast growth factor 23 (R176Q) delineate a putative role for parathyroid hormone in renal phosphate wasting disorders
XY Bai et al.
ENDOCRINOLOGY (2004)
Regulation of fibroblastic growth factor 23 expression but not degradation by PHEX
SG Liu et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Mutant FGF-23 responsible for autosomal dominant hypophosphatemic, rickets is resistant to proteolytic cleavage and causes hypophosphatemia in vivo
T Shimada et al.
ENDOCRINOLOGY (2002)
Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia
T Shimada et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2001)
Identification of a novel fibroblast growth factor, FGF-23, preferentially expressed in the ventrolateral thalamic nucleus of the brain
T Yamashita et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2000)
Genetic ablation of parathyroid glands reveals another source of parathyroid hormone
T Günther et al.
NATURE (2000)
Molecular mechanisms of secondary hyperparathyroidism
J Silver
NEPHROLOGY DIALYSIS TRANSPLANTATION (2000)
Share Paper
