Journal
NUCLEUS
Volume 6, Issue 5, Pages 394-407Publisher
TAYLOR & FRANCIS INC
DOI: 10.1080/19491034.2015.1084453
Keywords
AFM; cardiomyopathy; cardiomyocytes; cell physiology; lamin A/C; relaxation force test; Young Modulus
Categories
Funding
- Foreman-Casali Foundation, Trieste (Italy)
- NIH (USA) [UL1 RR025780, TR001082, RO1 HL69071, RO1 HL116906, K23 JL067915, 1R01HL109209-01A1]
- MISE-ICE-CRUI [99]
- FVG Region (Italy) [LR 26/2005 Art. 23]
- Kevin Kauffman Endowed Chair of Cardiology
- Foundation Leducq
- Transatlantic Network of Excellence [14-CVD 03]
Ask authors/readers for more resources
Previous investigations suggested that lamin A/C gene (LMNA) mutations, which cause a variety of human diseases including muscular dystrophies and cardiomyopathies, alter the nuclear mechanical properties. We hypothesized that biomechanical changes may extend beyond the nucleus.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available
Share Paper
