4.6 Article

Dystrophic neurites express C9orf72 in Alzheimer's disease brains

Related references

Note: Only part of the references are listed.
Article Clinical Neurology

Pattern of ubiquilin pathology in ALS and FTLD indicates presence of C9ORF72 hexanucleotide expansion

Johannes Brettschneider et al.

ACTA NEUROPATHOLOGICA (2012)

Article Clinical Neurology

The clinical and pathological phenotype of C9ORF72 hexanucleotide repeat expansions

Javier Simon-Sanchez et al.

BRAIN (2012)

Article Clinical Neurology

Immunohistochemical characterization of γ-secretase activating protein expression in Alzheimer's disease brains

J. Satoh et al.

NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY (2012)

Letter Medicine, General & Internal

Repeat Expansion in C9ORF72 in Alzheimer's Disease

Elisa Majounie et al.

NEW ENGLAND JOURNAL OF MEDICINE (2012)

Review Neurosciences

Emerging role of p62/sequestosome-1 in the pathogenesis of Alzheimer's disease

Antero Salminen et al.

PROGRESS IN NEUROBIOLOGY (2012)

Article Clinical Neurology

SQSTM1 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis

Faisal Fecto et al.

ARCHIVES OF NEUROLOGY (2011)

Article Multidisciplinary Sciences

Mutations in UBQLN2 cause dominant X-linked juvenile and adult-onset ALS and ALS/dementia

Han-Xiang Deng et al.

NATURE (2011)

Article

Primary Central Nervous System Lymphoma

Elizabeth R. Gerstner et al.

ARCHIVES OF NEUROLOGY (2010)

Article Biochemistry & Molecular Biology

The hPLIC proteins may provide a link between the ubiquitination machinery and the proteasome

MF Kleijnen et al.

MOLECULAR CELL (2000)