Related references
Note: Only part of the references are listed.The inactivation domain of STIM1 is functionally coupled with the Orai1 pore to enable Ca2+-dependent inactivation
Franklin M. Mullins et al.
JOURNAL OF GENERAL PHYSIOLOGY (2016)
Microenvironment acidity as a major determinant of tumor chemoresistance: Proton pump inhibitors (PPIs) as a novel therapeutic approach
Sophie Taylor et al.
DRUG RESISTANCE UPDATES (2015)
Identification of key amino acid residues responsible for internal and external pH sensitivity of Orai1/STIM1 channels
Hiroto Tsujikawa et al.
SCIENTIFIC REPORTS (2015)
Regulation of endogenous and heterologous Ca2+ release-activated Ca2+ currents by pH
Andreas Beck et al.
CELL CALCIUM (2014)
The chemistry, physiology and pathology of pH in cancer
Pawel Swietach et al.
PHILOSOPHICAL TRANSACTIONS OF THE ROYAL SOCIETY B-BIOLOGICAL SCIENCES (2014)
The effects of pH on wound healing, biofilms, and antimicrobial efficacy
Steven L. Percival et al.
WOUND REPAIR AND REGENERATION (2014)
Glu106 in the Orai1 pore contributes to fast Ca2+-dependent inactivation and pH dependence of Ca2+ release-activated Ca2+ (CRAC) current
Nathan R. Scrimgeour et al.
BIOCHEMICAL JOURNAL (2012)
Cooperativeness of Orai Cytosolic Domains Tunes Subtype-specific Gating
Irene Frischauf et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
Hypoxia-induced Acidosis Uncouples the STIM-Orai Calcium Signaling Complex
Salvatore Mancarella et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2011)
Protons and Ca2+: Ionic Allies in Tumor Progression?
Maike Glitsch
PHYSIOLOGY (2011)
Stoichiometric requirements for trapping and gating of Ca2+ release-activated Ca2+ (CRAC) channels by stromal interaction molecule 1 (STIM1)
Paul J. Hoover et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2011)
Cytosolic Ca2+ Buffers
Beat Schwaller
COLD SPRING HARBOR PERSPECTIVES IN BIOLOGY (2010)
Properties of Orai1 mediated store-operated current depend on the expression levels of STIM1 and Orai1 proteins
N. Scrimgeour et al.
JOURNAL OF PHYSIOLOGY-LONDON (2009)
STIM1 and calmodulin interact with Orai1 to induce Ca2+-dependent inactivation of CRAC channels
Franklin M. Mullins et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)
Activation mechanism for CRAC current and store-operated Ca2+ entry -: Calcium influx factor and Ca2+-independent phospholipase A2β-mediated pathway
Peter Csutora et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
Focusing of the electrostatic potential at EF-hands of calbindin D9k:: Titration of acidic residues
T Kesvatera et al.
PROTEINS-STRUCTURE FUNCTION AND BIOINFORMATICS (2001)
Potentiation and inhibition of Ca2+ release-activated Ca2+ channels by 2-aminoethyldiphenyl borate (2-APB) occurs independently of IP3 receptors
M Prakriya et al.
JOURNAL OF PHYSIOLOGY-LONDON (2001)
Share Paper
