Journal
CANCER DISCOVERY
Volume 3, Issue 1, Pages 24-26Publisher
AMER ASSOC CANCER RESEARCH
DOI: 10.1158/2159-8290.CD-12-0521
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Funding
- NATIONAL CANCER INSTITUTE [T32CA071341] Funding Source: NIH RePORTER
- NCI NIH HHS [T32 CA071341] Funding Source: Medline
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Although the functional interplay between mutant and wild-type Ras in driving tumor initiation and growth has been described, a clear picture of the precise ramifications and mechanisms of this association remains elusive, sometimes with conflicting conclusions. A report in this issue of Cancer Discovery tackles this question, which may have important implications for therapeutic strategies to block mutant Ras for cancer treatment. Cancer Discov; 3(1); 24-6. (C) 2012 AACR.
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