Journal
WEED TECHNOLOGY
Volume 26, Issue 1, Pages 156-160Publisher
WEED SCI SOC AMER
DOI: 10.1614/WT-D-11-00096.1
Keywords
ALS gene sequencing; ALS-inhibitor resistance; herbicide resistance; target-site mutation
Categories
Funding
- Alberta Crop Industry Development Fund Ltd. (ACIDF)
- Arysta LifeScience Canada Inc.
- BASF Canada Inc.
- Bayer CropScience Canada
- Dow AgroSciences Canada Inc.
- E. I. duPont Canada Co.
- Gowan Canada
- Monsanto Canada Inc.
- Nufarm Agriculture Inc.
- Syngenta Crop Protection Canada, Inc.
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Wild buckwheat is the most abundant broadleaf weed across the Prairie region of western Canada. Acetolactate synthase (ALS)-inhibiting herbicides are commonly used to control this species and other broadleaf weeds in cereal crops. A field survey in Alberta in 2007 identified a single population that was putatively resistant to ALS-inhibiting herbicides. In herbicide resistance screening in the greenhouse, all F-1 progeny tested were resistant to the ALS-inhibiting herbicides thifensulfuron/tribenuron, a sulfonylurea herbicide, or florasulam, a triazolopyrimidine herbicide; dose response of shoot biomass indicated the population was 10- and 20-fold less sensitive to thifensulfuron/tribenuron and florasulam, respectively, than a susceptible control population. ALS gene sequencing of 24 F1 progeny indicated that the Trp(574)Leu target-site mutation was responsible for conferring ALS-inhibitor resistance in this biotype, the first global report of ALS-inhibitor resistance for this species. Because this mutation typically endows high-level resistance across all five ALS-inhibitor classes, this wild buckwheat biotype may only be controlled by a different site-of-action herbicide.
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